Liang-Wei Gong

Transient forebrain ischemia induces persistent hyperactivity of large conductance Ca2+-activated potassium channels via oxidation modulation in rat hippocampal CA1 pyramidal neurons.

Involvement of actin cytoskeleton in modulation of Ca(2+)-activated K(+) channels from rat hippocampal CA1 pyramidal neurons.

Intracellular patch electrochemistry: regulation of cytosolic catecholamines in chromaffin cells.

Secretory vesicles membrane area is regulated in tandem with quantal size in chromaffin cells.

Phosphatidylinositol phosphate kinase type I gamma regulates dynamics of large dense-core vesicle fusion.

Patch amperometry: high-resolution measurements of single-vesicle fusion and release.

A selective activity-dependent requirement for dynamin 1 in synaptic vesicle endocytosis.

Exocytotic catecholamine release is not associated with cation flux through channels in the vesicle membrane but Na+ influx through the fusion pore.

The role of the C terminus of the SNARE protein SNAP-25 in fusion pore opening and a model for fusion pore mechanics.

Regulation of postsynaptic AMPA responses by synaptojanin 1.

Automatic detection of large dense-core vesicles in secretory cells and statistical analysis of their intracellular distribution.

Phosphatidylinositol-4-phosphate 5-kinases and phosphatidylinositol 4,5-bisphosphate synthesis in the brain.

Synaptotagmin 1 is necessary for the Ca2+ dependence of clathrin-mediated endocytosis.

Tenuigenin treatment improves behavioral Y-maze learning by enhancing synaptic plasticity in mice.

Effects of cordycepin on Y-maze learning task in mice.

Actin polymerization does not provide direct mechanical forces for vesicle fission during clathrin-mediated endocytosis.