Maureen Horton

NF-kappa B activation mediates the cross-talk between extracellular matrix and interferon-gamma (IFN-gamma) leading to enhanced monokine induced by IFN-gamma (MIG) expression in macrophages.

Pulmonary alveolar proteinosis.

The novel cyclophilin binding compound, sanglifehrin A, disassociates G1 cell cycle arrest from tolerance induction.

Identification of the molecular mechanism by which TLR ligation and IFN-gamma synergize to induce MIG.

Rheumatoid arthritis associated interstitial lung disease.

Egr-2 and Egr-3 are negative regulators of T cell activation.

Threat matrix: low-molecular-weight hyaluronan (HA) as a danger signal.

Differential regulation of hyaluronan-induced IL-8 and IP-10 in airway epithelial cells.

Hyaluronan fragments act as an endogenous danger signal by engaging TLR2.

Cutting Edge: TCR-induced NAB2 enhances T cell function by coactivating IL-2 transcription.

Ambient particulate matter directs nonclassic dendritic cell activation and a mixed TH1/TH2-like cytokine response by naive CD4+ T cells.

A clinician's guide to the diagnosis and treatment of interstitial lung diseases.

A2A receptor signaling promotes peripheral tolerance by inducing T-cell anergy and the generation of adaptive regulatory T cells.

Advanced visualization of airways with 64-MDCT: 3D mapping and virtual bronchoscopy.

Respiratory CSI: diagnosing occupationally related lung disease.

Sleep quality and health-related quality of life in idiopathic pulmonary fibrosis.

The adenosine a2a receptor inhibits matrix-induced inflammation in a novel fashion.

Anti-oxidant inhibition of hyaluronan fragment-induced inflammatory gene expression.

Ascertainment of collagen vascular disease in patients presenting with interstitial lung disease.

Quieting T cells with Slfn2.

Hyaluronan fragments promote inflammation by down-regulating the anti-inflammatory A2a receptor.

Application of carbon monoxide diffusing capacity in the mouse lung.

The kinase mTOR regulates the differentiation of helper T cells through the selective activation of signaling by mTORC1 and mTORC2.

Rheumatoid arthritis-associated interstitial lung disease: diagnostic dilemma.

Enhancement of tumor immunotherapy by deletion of the A2A adenosine receptor.

Regulation of immune responses by mTOR.

Pulmonary vaccination as a novel treatment for lung fibrosis.

Severe pulmonary hypertension in idiopathic nonspecific interstitial pneumonia.

Thalidomide for the treatment of cough in idiopathic pulmonary fibrosis: a randomized trial.

Regulation of CD4⁺ and CD8⁺ effector responses by Sprouty-1.

Macrophage A2A adenosinergic receptor modulates oxygen-induced augmentation of murine lung injury.

Validation of the Cough Quality-of-Life Questionnaire in patients with idiopathic pulmonary fibrosis.

Author reply: To PMID 22986377.

Hyaluronan fragments induce IFNβ via a novel TLR4-TRIF-TBK1-IRF3-dependent pathway.

Source of biomass cooking fuel determines pulmonary response to household air pollution.

Low molecular weight hyaluronan activates cytosolic phospholipase A2α and eicosanoid production in monocytes and macrophages.

Egr3 induces a Th17 response by promoting the development of γδ T cells.

An Fc domain protein-small molecule conjugate as an enhanced immunomodulator.

Interstitial lung disease in patients with rheumatoid arthritis: spontaneous and drug induced.

H2S relaxes isolated human airway smooth muscle cells via the sarcolemmal K(ATP) channel.

Immunological priming requires regulatory T cells and IL-10-producing macrophages to accelerate resolution from severe lung inflammation.

The AGC kinase SGK1 regulates TH1 and TH2 differentiation downstream of the mTORC2 complex.

Pulmonary delivery of docosahexaenoic acid mitigates bleomycin-induced pulmonary fibrosis.

A mouse model of chronic idiopathic pulmonary fibrosis.

An in situ, in vivo murine model for the study of laryngotracheal stenosis.

Lovastatin Inhibits Low Molecular Weight Hyaluronan Induced Chemokine Expression via LFA-1 and Decreases Bleomycin-Induced Pulmonary Fibrosis.