Lan Mao

Heart size-independent analysis of myocardial function in murine pressure overload hypertrophy.

Pressure overload selectively up-regulates Ca2+/calmodulin-dependent protein kinase II in vivo.

Transgenic overexpression of the Ca2+-binding protein S100A1 in the heart leads to increased in vivo myocardial contractile performance.

Inhibition of receptor-localized PI3K preserves cardiac beta-adrenergic receptor function and ameliorates pressure overload heart failure.

Role for thromboxane receptors in angiotensin-II-induced hypertension.

Cardiac hypertrophy and sudden death in mice with a genetically clamped renin transgene.

Enhanced postischemic functional recovery in CYP2J2 transgenic hearts involves mitochondrial ATP-sensitive K+ channels and p42/p44 MAPK pathway.

Estrogen receptor-beta mediates male-female differences in the development of pressure overload hypertrophy.

cTnT1, a cardiac troponin T isoform, decreases myofilament tension and affects the left ventricular pressure waveform.

An essential role for mitochondrial aldehyde dehydrogenase in nitroglycerin bioactivation.

Prostacyclin protects against elevated blood pressure and cardiac fibrosis.

Competitive displacement of phosphoinositide 3-kinase from beta-adrenergic receptor kinase-1 improves postinfarction adverse myocardial remodeling.

Intermittent pressure overload triggers hypertrophy-independent cardiac dysfunction and vascular rarefaction.

Altered blood pressure responses and normal cardiac phenotype in ACE2-null mice.

Oxidative phenotype protects myofibers from pathological insults induced by chronic heart failure in mice.

Cardiac micro-computed tomography for morphological and functional phenotyping of muscle LIM protein null mice.

Increased myocardial contractility and enhanced exercise function in transgenic mice overexpressing either adenylyl cyclase 5 or 8.

Enhanced calcium cycling and contractile function in transgenic hearts expressing constitutively active G alpha o* protein.

Effect of the nitric oxide donor V-PYRRO/NO on portal pressure and sinusoidal dynamics in normal and cirrhotic mice.

A role for the thromboxane receptor in L-NAME hypertension.

Discordant on/off switching of gene expression in myocytes during cardiac hypertrophy in vivo.

Beta1-adrenergic receptors stimulate cardiac contractility and CaMKII activation in vivo and enhance cardiac dysfunction following myocardial infarction.

Tnni3k modifies disease progression in murine models of cardiomyopathy.

TRPC1 channels are critical for hypertrophic signaling in the heart.

Exogenously administered secreted frizzled related protein 2 (Sfrp2) reduces fibrosis and improves cardiac function in a rat model of myocardial infarction.

β-Arrestin mediates oxytocin receptor signaling, which regulates uterine contractility and cellular migration.

X-Linked Inhibitor of Apoptosis Protein-Mediated Attenuation of Apoptosis, Using a Novel Cardiac-Enhanced Adeno-Associated Viral Vector.

Host acyl-CoA binding protein regulates replication complex assembly and activity of a positive-strand RNA virus.

Diabetic kidney disease in FVB/NJ Akita mice: temporal pattern of kidney injury and urinary nephrin excretion.

p53 functions in endothelial cells to prevent radiation-induced myocardial injury in mice.

β-Arrestin-biased AT1R stimulation promotes cell survival during acute cardiac injury.

Overexpression of TNNI3K, a cardiac-specific MAPKKK, promotes cardiac dysfunction.

CCR2 deficiency, dysregulation of Notch signaling, and spontaneous pulmonary arterial hypertension.

Normal cardiac function in mice with supraphysiological cardiac creatine levels.

β-arrestin1-biased β1-adrenergic receptor signaling regulates microRNA processing.

Anatomical and functional imaging of myocardial infarction in mice using micro-CT and eXIA 160 contrast agent.

Atm deletion with dual recombinase technology preferentially radiosensitizes tumor endothelium.

Neuroimaging effects of 1 Hz right temporoparietal rTMS on normal auditory processing: implications for clinical hallucination treatment paradigms.

Circulating Exosomes Induced by Cardiac Pressure Overload Contain Functional Angiotensin II Type 1 Receptors.

The role of β-arrestin2-dependent signaling in thoracic aortic aneurysm formation in a murine model of Marfan Syndrome.

Lethal Cardiomyopathy in Mice Lacking Transferrin Receptor in the Heart.

Enhanced Uterine Contractility and Stillbirth in Mice Lacking G Protein-Coupled Receptor Kinase 6 (GRK6): Implications for Oxytocin Receptor Desensitization.