Sudhanshu Bhushan

Uropathogenic Escherichia coli block MyD88-dependent and activate MyD88-independent signaling pathways in rat testicular cells.

Testicular innate immune defense against bacteria.

Testicular infection: microorganisms, clinical implications and host-pathogen interaction.

Immunoprivileged sites: the testis.

Testosterone replacement effectively inhibits the development of experimental autoimmune orchitis in rats: evidence for a direct role of testosterone on regulatory T cell expansion.

Uropathogenic E. coli induce different immune response in testicular and peritoneal macrophages: implications for testicular immune privilege.

Necrosis is the dominant cell death pathway in uropathogenic Escherichia coli elicited epididymo-orchitis and is responsible for damage of rat testis.

Structural and functional integrity of spermatozoa is compromised as a consequence of acute uropathogenic E. coli-associated epididymitis.

Uropathogenic Escherichia coli modulates innate immunity to suppress Th1-mediated inflammatory responses during infectious epididymitis.

H3K79 methylation: a new conserved mark that accompanies H4 hyperacetylation prior to histone-to-protamine transition in Drosophila and rat.

Targeting high mobility group box protein 1 ameliorates testicular inflammation in experimental autoimmune orchitis.

Influence of Testosterone on Inflammatory Response in Testicular Cells and Expression of Transcription Factor Foxp3 in T Cells.

Differential activation of inflammatory pathways in testicular macrophages provides a rationale for their subdued inflammatory capacity.

Uropathogenic Escherichia coli Epigenetically Manipulate Host Cell Death Pathways.