J. Ashot Kozak

Distinct properties of CRAC and MIC channels in RBL cells.

A transient receptor potential channel expressed in taste receptor cells.

MIC channels are inhibited by internal divalent cations but not ATP.

Polyvalent cations as permeant probes of MIC and TRPM7 pores.

Channel function is dissociated from the intrinsic kinase activity and autophosphorylation of TRPM7/ChaK1.

STIM1, an essential and conserved component of store-operated Ca2+ channel function.

STIM1 is a Ca2+ sensor that activates CRAC channels and migrates from the Ca2+ store to the plasma membrane.

Charge screening by internal pH and polyvalent cations as a mechanism for activation, inhibition, and rundown of TRPM7/MIC channels.

Soluble amyloid oligomers increase bilayer conductance by altering dielectric structure.

Orai1 and STIM1 move to the immunological synapse and are up-regulated during T cell activation.

Store-dependent and -independent modes regulating Ca2+ release-activated Ca2+ channel activity of human Orai1 and Orai3.

Detailed examination of Mg2+ and pH sensitivity of human TRPM7 channels.

Sensitivity of TRPM7 channels to Mg2+ characterized in cell-free patches of Jurkat T lymphocytes.

Effect of synthetic aβ peptide oligomers and fluorinated solvents on Kv1.3 channel properties and membrane conductance.

2-aminoethyl diphenyl borinate (2-APB) inhibits TRPM7 channels through an intracellular acidification mechanism.

Inactivation of TRPM7 kinase activity does not impair its channel function in mice.