Tino Hochepied

Mice lacking alpha(2)-macroglobulin show an increased host defense against Gram-negative bacterial sepsis, but are more susceptible to endotoxic shock.

Alpha(1)-acid glycoprotein: an acute phase protein with inflammatory and immunomodulating properties.

Breaking the species barrier: derivation of germline-competent embryonic stem cells from Mus spretus x C57BL/6 hybrids.

Response of TNF-hyporesponsive SPRET/Ei mice in models of inflammatory disorders.

Exogenous alpha-1-acid glycoprotein protects against renal ischemia-reperfusion injury by inhibition of inflammation and apoptosis.

The wild-derived inbred mouse strain SPRET/Ei is resistant to LPS and defective in IFN-beta production.

Caspase-14 protects against epidermal UVB photodamage and water loss.

Cop1 constitutively regulates c-Jun protein stability and functions as a tumor suppressor in mice.

Efficient and user-friendly pluripotin-based derivation of mouse embryonic stem cells.

The ROSA26-iPSC mouse: a conditional, inducible, and exchangeable resource for studying cellular (De)differentiation.

Mice overexpressing β-1,4-Galactosyltransferase I are resistant to TNF-induced inflammation and DSS-induced colitis.

An ER-directed gelsolin nanobody targets the first step in amyloid formation in a gelsolin amyloidosis mouse model.

Cardiovascular and pharmacological implications of haem-deficient NO-unresponsive soluble guanylate cyclase knock-in mice.

p120 Catenin-Mediated Stabilization of E-Cadherin Is Essential for Primitive Endoderm Specification.

Elevated ΔNp63α Levels Facilitate Epidermal and Biliary Oncogenic Transformation.

Characterization of New Transgenic Mouse Models for Two Charcot-Marie-Tooth-Causing HspB1 Mutations using the Rosa26 Locus.

NLRP2 controls age-associated maternal fertility.

Familial Mediterranean fever mutations lift the obligatory requirement for microtubules in Pyrin inflammasome activation.

Overexpression of Glucocorticoid-induced Leucine Zipper (GILZ) increases susceptibility to Imiquimod-induced psoriasis and involves cutaneous activation of TGF-β1.

DMRT5 Together with DMRT3 Directly Controls Hippocampus Development and Neocortical Area Map Formation.