Julia V. Busik

Glucose-induced activation of glucose uptake in cells from the inner and outer blood-retinal barrier.

Are diabetic neuropathy, retinopathy and nephropathy caused by hyperglycemic exclusion of dehydroascorbate uptake by glucose transporters?

Regulation of hepatic GLUT8 expression in normal and diabetic models.

Dyslipidemia, but not hyperglycemia, induces inflammatory adhesion molecules in human retinal vascular endothelial cells.

Anti-inflammatory effect of docosahexaenoic acid on cytokine-induced adhesion molecule expression in human retinal vascular endothelial cells.

Regulation of hepatic fatty acid elongase and desaturase expression in diabetes and obesity.

Inhibition of cytokine signaling in human retinal endothelial cells through modification of caveolae/lipid rafts by docosahexaenoic acid.

Hyperglycemia-induced reactive oxygen species toxicity to endothelial cells is dependent on paracrine mediators.

Carbon monoxide and nitric oxide mediate cytoskeletal reorganization in microvascular cells via vasodilator-stimulated phosphoprotein phosphorylation: evidence for blunted responsiveness in diabetes.

Archived unfrozen neonatal blood spots are amenable to quantitative gene expression analysis.

Differential regulation of high glucose-induced glyceraldehyde-3-phosphate dehydrogenase nuclear accumulation in Müller cells by IL-1beta and IL-6.

Novel mechanism for obesity-induced colon cancer progression.

Complementary precursor ion and neutral loss scan mode tandem mass spectrometry for the analysis of glycerophosphatidylethanolamine lipids from whole rat retina.

Global analysis of retina lipids by complementary precursor ion and neutral loss mode tandem mass spectrometry.

Remodeling of retinal Fatty acids in an animal model of diabetes: a decrease in long-chain polyunsaturated fatty acids is associated with a decrease in fatty acid elongases Elovl2 and Elovl4.

Diabetic retinopathy is associated with bone marrow neuropathy and a depressed peripheral clock.

Inhibition of cytokine signaling in human retinal endothelial cells through downregulation of sphingomyelinases by docosahexaenoic acid.

Analysis of Retina and Erythrocyte Glycerophospholipid Alterations in a Rat Model of Type 1 Diabetes.

The unconventional role of acid sphingomyelinase in regulation of retinal microangiopathy in diabetic human and animal models.

Free insulin-like growth factor binding protein-3 (IGFBP-3) reduces retinal vascular permeability in association with a reduction of acid sphingomyelinase (ASMase).

Evaluation of sex-specific gene expression in archived dried blood spots (DBS).

Per2 mutation recapitulates the vascular phenotype of diabetes in the retina and bone marrow.

N-3 polyunsaturated Fatty acids prevent diabetic retinopathy by inhibition of retinal vascular damage and enhanced endothelial progenitor cell reparative function.

Examining the role of lipid mediators in diabetic retinopathy.

CNS inflammation and bone marrow neuropathy in type 1 diabetes.

Dicer expression exhibits a tissue-specific diurnal pattern that is lost during aging and in diabetes.

Aldose reductase meets histone acetylation: a new role for an old player.

Effect of reduced retinal VLC-PUFA on rod and cone photoreceptors.

Changes in the daily rhythm of lipid metabolism in the diabetic retina.

Regulation of retinal inflammation by rhythmic expression of MiR-146a in diabetic retina.

A monophasic extraction strategy for the simultaneous lipidome analysis of polar and nonpolar retina lipids.

Wnting out ocular neovascularization: using nanoparticle delivery of very-low density lipoprotein receptor extracellular domain as Wnt pathway inhibitor in the retina.

Ataxia Telangiectasia Mutated Dysregulation Results in Diabetic Retinopathy.

Role of Acid Sphingomyelinase in Shifting the Balance Between Proinflammatory and Reparative Bone Marrow Cells in Diabetic Retinopathy.

Imbalances in Mobilization and Activation of Pro-Inflammatory and Vascular Reparative Bone Marrow-Derived Cells in Diabetic Retinopathy.

Dual Anti-Inflammatory and Anti-Angiogenic Action of miR-15a in Diabetic Retinopathy.

Effect of storage time on gene expression data acquired from unfrozen archived newborn blood spots.

Interplay between Endothelial Cell Cytoskeletal Rigidity and Plasma Membrane Fluidity.

Conditional Deletion of Bmal1 Accentuates Microvascular and Macrovascular Injury.

Tumor Necrosis Factor Alpha (TNF-α) Disrupts Kir4.1 Channel Expression Resulting in Müller Cell Dysfunction in the Retina.

The role of dyslipidemia in diabetic retinopathy.

The Mechanism of Diabetic Retinopathy Pathogenesis Unifying Key Lipid Regulators, Sirtuin 1 and Liver X Receptor.