Diane Barber

Ion transport proteins anchor and regulate the cytoskeleton.

Somatostatin, acting at receptor subtype 1, inhibits Rho activity, the assembly of actin stress fibers, and cell migration.

Cell migration requires both ion translocation and cytoskeletal anchoring by the Na-H exchanger NHE1.

Conserved motifs in somatostatin, D2-dopamine, and alpha 2B-adrenergic receptors for inhibiting the Na-H exchanger, NHE1.

Na-H exchange-dependent increase in intracellular pH times G2/M entry and transition.

Expression profile of genes regulated by activity of the Na-H exchanger NHE1.

Na(+)/H(+) exchanger NHE1 as plasma membrane scaffold in the assembly of signaling complexes.

A developmentally regulated Na-H exchanger in Dictyostelium discoideum is necessary for cell polarity during chemotaxis.

The Nck-interacting kinase phosphorylates ERM proteins for formation of lamellipodium by growth factors.

Intracellular pH sensors: design principles and functional significance.

Pilot study of regional, hepatic intra-arterial paclitaxel in patients with breast carcinoma metastatic to the liver.

Beyond ion translocation: structural functions of the sodium-hydrogen exchanger isoform-1.

Positive feedback between Cdc42 activity and H+ efflux by the Na-H exchanger NHE1 for polarity of migrating cells.

Phosphorylation of the Arp2/3 complex is necessary to nucleate actin filaments.

Cofilin is a pH sensor for actin free barbed end formation: role of phosphoinositide binding.

Mislocalized scaffolding by the Na-H exchanger NHE1 dominantly inhibits fibronectin production and TGF-beta activation.

The sodium-hydrogen exchanger NHE1 is an Akt substrate necessary for actin filament reorganization by growth factors.

Expression of actin-interacting protein 1 suppresses impaired chemotaxis of Dictyostelium cells lacking the Na+-H+ exchanger NHE1.

Nuclear-localized calcineurin homologous protein CHP1 interacts with upstream binding factor and inhibits ribosomal RNA synthesis.

A human MAP kinase interactome.

Dysregulated pH: a perfect storm for cancer progression.

Outcome analyses after the first admission to an intensive care unit in patients with advanced cancer referred to a phase I clinical trials program.

Dynamic actin remodeling during epithelial-mesenchymal transition depends on increased moesin expression.

Phosphorylation of the Arp2 subunit relieves auto-inhibitory interactions for Arp2/3 complex activation.