Anna Maisa

The role of myristoylation in the membrane association of the Lassa virus matrix protein Z.

Inhibition of Lassa virus glycoprotein cleavage and multicycle replication by site 1 protease-adapted alpha(1)-antitrypsin variants.

Characterization of Lassa virus glycoprotein oligomerization and influence of cholesterol on virus replication.

Viral protein determinants of Lassa virus entry and release from polarized epithelial cells.

HIV infection induces age-related changes to monocytes and innate immune activation in young men that persist despite combination antiretroviral therapy.

Endothelial cell activation promotes foam cell formation by monocytes following transendothelial migration in an in vitro model.

Aging is associated with chronic innate immune activation and dysregulation of monocyte phenotype and function.

Associations between surface markers on blood monocytes and carotid atherosclerosis in HIV-positive individuals.

Increased glucose metabolic activity is associated with CD4+ T-cell activation and depletion during chronic HIV infection.

Viremic and Virologically Suppressed HIV Infection Increases Age-Related Changes to Monocyte Activation Equivalent to 12 and 4 Years of Aging, Respectively.

Monocytes from HIV-infected individuals show impaired cholesterol efflux and increased foam cell formation after transendothelial migration.

Epidemiological investigation and case-control study: a Legionnaires' disease outbreak associated with cooling towers in Warstein, Germany, August-September 2013.

Ex vivo foam cell formation is enhanced in monocytes from older individuals by both extrinsic and intrinsic mechanisms.

Genetic polymorphisms in the CD14 gene are associated with monocyte activation and carotid intima-media thickness in HIV-infected patients on antiretroviral therapy.