Yuval Rinkevich

Localized hepatic lobular regeneration by central-vein-associated lineage-restricted progenitors.

Two succeeding fibroblastic lineages drive dermal development and the transition from regeneration to scarring.

Mesothelial to mesenchyme transition as a major developmental and pathological player in trunk organs and their cavities.

Defining Skin Fibroblastic Cell Types Beyond CD90.

Surgical adhesions in mice are derived from mesothelial cells and can be targeted by antibodies against mesothelial markers.

Neutrophil and monocyte kinetics play critical roles in mouse peritoneal adhesion formation.

Local and transient inhibition of p21 expression ameliorates age-related delayed wound healing.

Patch repair of deep wounds by mobilized fascia.

Scars or Regeneration?-Dermal Fibroblasts as Drivers of Diverse Skin Wound Responses.

Post-surgical adhesions are triggered by calcium-dependent membrane bridges between mesothelial surfaces.

Injury triggers fascia fibroblast collective cell migration to drive scar formation through N-cadherin.

Connexin43 gap junction drives fascia mobilization and repair of deep skin wounds.

Distinct fibroblasts in scars and regeneration.

Fibroblasts as confederates of the immune system.

Cutting into wound repair.

Furnishing Wound Repair by the Subcutaneous Fascia.

Converting fibroblastic fates leads to wound healing without scar.

Epidermal-Derived Hedgehog Signaling Drives Mesenchymal Proliferation during Digit Tip Regeneration.

Milk exosomes-mediated miR-31-5p delivery accelerates diabetic wound healing through promoting angiogenesis.

Calcium ion cross-linked sodium alginate hydrogels containing deferoxamine and copper nanoparticles for diabetic wound healing.

Neutrophils direct preexisting matrix to initiate repair in damaged tissues.

Genital Wound Repair and Scarring.