Anthony N. Imbalzano

Phosphorylation of linker histones regulates ATP-dependent chromatin remodeling enzymes.

The myogenic basic helix-loop-helix family of transcription factors shows similar requirements for SWI/SNF chromatin remodeling enzymes during muscle differentiation in culture.

Unfolding heterochromatin for replication.

Repositioning of muscle-specific genes relative to the periphery of SC-35 domains during skeletal myogenesis.

Transcriptional compensation for loss of an allele of the Ini1 tumor suppressor.

Functional properties of ATP-dependent chromatin remodeling enzymes.

Loss of the INI1 tumor suppressor does not impair the expression of multiple BRG1-dependent genes or the assembly of SWI/SNF enzymes.

BRCA1 interacts with dominant negative SWI/SNF enzymes without affecting homologous recombination or radiation-induced gene activation of p21 or Mdm2.

Temporal recruitment of transcription factors and SWI/SNF chromatin-remodeling enzymes during adipogenic induction of the peroxisome proliferator-activated receptor gamma nuclear hormone receptor.

p38 pathway targets SWI-SNF chromatin-remodeling complex to muscle-specific loci.

Role for Nhp6, Gcn5, and the Swi/Snf complex in stimulating formation of the TATA-binding protein-TFIIA-DNA complex.

Inducible changes in cell size and attachment area due to expression of a mutant SWI/SNF chromatin remodeling enzyme.

SWI/SNF chromatin remodeling complex is obligatory for BMP2-induced, Runx2-dependent skeletal gene expression that controls osteoblast differentiation.

Snf5 tumor suppressor couples chromatin remodeling, checkpoint control, and chromosomal stability.

MyoD targets chromatin remodeling complexes to the myogenin locus prior to forming a stable DNA-bound complex.

Brg1, the ATPase subunit of the SWI/SNF chromatin remodeling complex, is required for myeloid differentiation to granulocytes.

Effects of HMGN1 on chromatin structure and SWI/SNF-mediated chromatin remodeling.

Chromatin domain activation via GATA-1 utilization of a small subset of dispersed GATA motifs within a broad chromosomal region.

Skeletal muscle specification by myogenin and Mef2D via the SWI/SNF ATPase Brg1.

HMGN1 is dispensable for myogenesis and adipogenesis.

Selective and antagonistic functions of SWI/SNF and Mi-2beta nucleosome remodeling complexes during an inflammatory response.

The ultrastructure of MCF-10A acini.

The microphthalmia-associated transcription factor requires SWI/SNF enzymes to activate melanocyte-specific genes.

Chromatin remodelling in mammalian differentiation: lessons from ATP-dependent remodellers.

Chromatin remodeling and transcriptional activity of the bone-specific osteocalcin gene require CCAAT/enhancer-binding protein beta-dependent recruitment of SWI/SNF activity.

Mutation of the SNF2 family member Chd2 affects mouse development and survival.

Functional interaction of the retinoblastoma and Ini1/Snf5 tumor suppressors in cell growth and pituitary tumorigenesis.

Mammalian SWI/SNF complexes facilitate DNA double-strand break repair by promoting gamma-H2AX induction.

The protein arginine methyltransferase Prmt5 is required for myogenesis because it facilitates ATP-dependent chromatin remodeling.

Interaction of papillomavirus E2 protein with the Brm chromatin remodeling complex leads to enhanced transcriptional activation.

Myogenin and the SWI/SNF ATPase Brg1 maintain myogenic gene expression at different stages of skeletal myogenesis.

Mitotic occupancy and lineage-specific transcriptional control of rRNA genes by Runx2.

Chromatin remodeling by SWI/SNF results in nucleosome mobilization to preferential positions in the rat osteocalcin gene promoter.

Deletion of p37Ing1 in mice reveals a p53-independent role for Ing1 in the suppression of cell proliferation, apoptosis, and tumorigenesis.

The Chd family of chromatin remodelers.

SWI/SNF chromatin remodeling ATPase Brm regulates the differentiation of early retinal stem cells/progenitors by influencing Brn3b expression and Notch signaling.

Phenotypic transcription factors epigenetically mediate cell growth control.

Comparative in silico analysis identifies bona fide MyoD binding sites within the Myocyte stress 1 gene promoter.

Loss of miRNA biogenesis induces p19Arf-p53 signaling and senescence in primary cells.

p37Ing1b regulates B-cell proliferation and cooperates with p53 to suppress diffuse large B-cell lymphomagenesis.

A mutation in the mouse Chd2 chromatin remodeling enzyme results in a complex renal phenotype.

Induction of TLR4-target genes entails calcium/calmodulin-dependent regulation of chromatin remodeling.

Distinct protein arginine methyltransferases promote ATP-dependent chromatin remodeling function at different stages of skeletal muscle differentiation.

Increasingly transformed MCF-10A cells have a progressively tumor-like phenotype in three-dimensional basement membrane culture.

The SWI/SNF chromatin remodeling complex regulates myocardin-induced smooth muscle-specific gene expression.

SWI/SNF-independent nuclease hypersensitivity and an increased level of histone acetylation at the P1 promoter accompany active transcription of the bone master gene Runx2.

Ectopic runx2 expression in mammary epithelial cells disrupts formation of normal acini structure: implications for breast cancer progression.

SWI/SNF chromatin remodeling enzyme ATPases promote cell proliferation in normal mammary epithelial cells.

Myogenic microRNA expression requires ATP-dependent chromatin remodeling enzyme function.

The human SWI/SNF complex associates with RUNX1 to control transcription of hematopoietic target genes.

An essential role for Dicer in adipocyte differentiation.

Chromatin accessibility and transcription factor binding at the PPARγ2 promoter during adipogenesis is protein kinase A-dependent.

Live cell imaging of the cancer-related transcription factor RUNX2 during mitotic progression.

Dicer is required for the formation of white but not brown adipose tissue.

The expression of myogenic microRNAs indirectly requires protein arginine methyltransferase (Prmt)5 but directly requires Prmt4.

Architectural genetic and epigenetic control of regulatory networks: compartmentalizing machinery for transcription and chromatin remodeling in nuclear microenvironments.

Versatility of PRMT5-induced methylation in growth control and development.

Isolation of nuclei from skeletal muscle satellite cells and myofibers for use in chromatin immunoprecipitation assays.

An improved restriction enzyme accessibility assay for analyzing changes in chromatin structure in samples of limited cell number.