Koh Nakata

Oligoclonal T cell expansions in pulmonary lymphoproliferative disorders: demonstration of the frequent occurrence of oligoclonal T cells in human immunodeficiency virus-related lymphoid interstitial pneumonia.

Genetic variants of human beta-defensin-1 and chronic obstructive pulmonary disease.

Maximal HIV-1 replication in alveolar macrophages during tuberculosis requires both lymphocyte contact and cytokines.

[The mechanism of HIV replication at the site of inflammation coinfected with HIV and M. tuberculosis].

Secondary pulmonary alveolar proteinosis associated with myelodysplastic syndrome.

High-affinity autoantibodies specifically eliminate granulocyte-macrophage colony-stimulating factor activity in the lungs of patients with idiopathic pulmonary alveolar proteinosis.

Pulmonary alveolar proteinosis.

Evaluation of choroidal perfusion of the new central macular area by dilution analysis of indocyanine green angiography after macular translocation.

BCL-6 mutations in pulmonary lymphoproliferative disorders: demonstration of an aberrant immunological reaction in HIV-related lymphoid interstitial pneumonia.

A case of idiopathic pulmonary alveolar proteinosis accompanied by T-cell receptor gene rearrangement in bronchoalveolar lavage fluid cells.

Pulmonary alveolar proteinosis.

Direct determination of MUC5B promoter haplotypes based on the method of single-strand conformation polymorphism and their statistical estimation.

Serum neutralizing capacity of GM-CSF reflects disease severity in a patient with pulmonary alveolar proteinosis successfully treated with inhaled GM-CSF.

Promoter analysis and aberrant expression of the MUC5B gene in diffuse panbronchiolitis.

[Molecular pathogenesis in tuberculosis complicated with AIDS].

Granulocyte-macrophage colony-stimulating factor and lung immunity in pulmonary alveolar proteinosis.

Propionibacterium acnes is the most common bacterium commensal in peripheral lung tissue and mediastinal lymph nodes from subjects without sarcoidosis.

Variations of the CFTR gene in the Hanoi-Vietnamese.

Epidemiological and clinical features of idiopathic pulmonary alveolar proteinosis in Japan.

Granulocyte-macrophage colony-stimulating factor inhalation therapy for patients with idiopathic pulmonary alveolar proteinosis: a pilot study; and long-term treatment with aerosolized granulocyte-macrophage colony-stimulating factor: a case report.

Why does the autoantibody against granulocyte-macrophage colony-stimulating factor cause lesions only in the lung?

Successful treatment of congenital pulmonary alveolar proteinosis with intravenous immunoglobulin G administration.

Serum antibody against granulocyte/macrophage colony-stimulating factor and KL-6 in idiopathic pulmonary alveolar proteinosis.

Mechanisms of polymorphonuclear neutrophil-mediated induction of HIV-1 replication in macrophages during pulmonary tuberculosis.

Expression of PU.1 and terminal differentiation of alveolar macrophages in newborn rats.

Anti-cytokine autoantibodies are ubiquitous in healthy individuals.

Characteristics of a large cohort of patients with autoimmune pulmonary alveolar proteinosis in Japan.

A combination therapy of whole lung lavage and GM-CSF inhalation in pulmonary alveolar proteinosis.

Granulocyte/macrophage colony-stimulating factor autoantibodies and myeloid cell immune functions in healthy individuals.

Reciprocal CD4+ T-cell balance of effector CD62Llow CD4+ and CD62LhighCD25+ CD4+ regulatory T cells in small cell lung cancer reflects disease stage.

Pulmonary alveolar proteinosis in a patient with Behcet's disease.

Characterization of human cultured periosteal sheets expressing bone-forming potential: in vitro and in vivo animal studies.

Granulocyte/macrophage-colony-stimulating factor autoantibodies and myeloid cell immune functions in healthy subjects.

Identification of MICA as a susceptibility gene for pulmonary Mycobacterium avium complex infection.

Expression of Th2-skewed pathology mediators in monocyte-derived type 2 of dendritic cells (DC2).

Comparative study of high-resolution CT findings between autoimmune and secondary pulmonary alveolar proteinosis.

Response: Granulocyte/macrophage colony-stimulating factor autoantibodies and myeloid cell immune functions in healthy persons.

Inhaled granulocyte/macrophage-colony stimulating factor as therapy for pulmonary alveolar proteinosis.

Pulmonary alveolar proteinosis associated with dust inhalation: not secondary but autoimmune?

Patient-derived granulocyte/macrophage colony-stimulating factor autoantibodies reproduce pulmonary alveolar proteinosis in nonhuman primates.

High avidity cytokine autoantibodies in health and disease: pathogenesis and mechanisms.

A cell-free assay to estimate the neutralizing capacity of granulocyte-macrophage colony-stimulating factor autoantibodies.

Adult-onset hereditary pulmonary alveolar proteinosis caused by a single-base deletion in CSF2RB.

Inflammation provoked by Mycoplasma pneumoniae extract: implications for combination treatment with clarithromycin and dexamethasone.

Efficacy and safety of sirolimus in lymphangioleiomyomatosis.

Vaccination with CD133(+) melanoma induces specific Th17 and Th1 cell-mediated antitumor reactivity against parental tumor.

[autoimmune pulmonary alveolar proteinosis and GM-CSF inhalation therapy].

Indium-tin oxide does not induce GM-CSF autoantibodies.

Direct evidence that GM-CSF inhalation improves lung clearance in pulmonary alveolar proteinosis.