J. Alexandra Rowe

Identification of a conserved Plasmodium falciparum var gene implicated in malaria in pregnancy.

Expanding the paradigms of placental malaria.

Short report: Positive correlation between rosetting and parasitemia in Plasmodium falciparum clinical isolates.

Nonimmune IgM, but not IgG binds to the surface of Plasmodium falciparum-infected erythrocytes and correlates with rosetting and severe malaria.

Plasmodium chabaudi: rosetting in a rodent malaria model.

A well-conserved Plasmodium falciparum var gene shows an unusual stage-specific transcript pattern.

CR1 Knops blood group alleles are not associated with severe malaria in the Gambia.

Nonspecific immunoglobulin M binding and chondroitin sulfate A binding are linked phenotypes of Plasmodium falciparum isolates implicated in malaria during pregnancy.

A human complement receptor 1 polymorphism that reduces Plasmodium falciparum rosetting confers protection against severe malaria.

Plasmodium falciparum: Rosettes do not protect merozoites from invasion-inhibitory antibodies.

Identification of Plasmodium falciparum var1CSA and var2CSA domains that bind IgM natural antibodies.

Low multiplication rates of African Plasmodium falciparum isolates and lack of association of multiplication rate and red blood cell selectivity with malaria virulence.

Virulence of malaria is associated with differential expression of Plasmodium falciparum var gene subgroups in a case-control study.

Expression of Plasmodium falciparum genes involved in erythrocyte invasion varies among isolates cultured directly from patients.

Transcribed var genes associated with placental malaria in Malawian women.

Differential var gene transcription in Plasmodium falciparum isolates from patients with cerebral malaria compared to hyperparasitaemia.

In vitro inhibition of Plasmodium falciparum rosette formation by Curdlan sulfate.

Invasion pathways and malaria severity in Kenyan Plasmodium falciparum clinical isolates.

Blood group O protects against severe Plasmodium falciparum malaria through the mechanism of reduced rosetting.

Platelet-mediated clumping of Plasmodium falciparum infected erythrocytes is associated with high parasitemia but not severe clinical manifestations of malaria in African children.

Identification of residues in the Cmu4 domain of polymeric IgM essential for interaction with Plasmodium falciparum erythrocyte membrane protein 1 (PfEMP1).

Experimental conditions affect the outcome of Plasmodium falciparum platelet-mediated clumping assays.

Adhesion of Plasmodium falciparum-infected erythrocytes to human cells: molecular mechanisms and therapeutic implications.

Putative DNA G-quadruplex formation within the promoters of Plasmodium falciparum var genes.

Blood groups and malaria: fresh insights into pathogenesis and identification of targets for intervention.

High levels of Plasmodium falciparum rosetting in all clinical forms of severe malaria in African children.

IgM, Fc mu Rs, and malarial immune evasion.

Immunisation with recombinant PfEMP1 domains elicits functional rosette-inhibiting and phagocytosis-inducing antibodies to Plasmodium falciparum.

Design of a variant surface antigen-supplemented microarray chip for whole transcriptome analysis of multiple Plasmodium falciparum cytoadherent strains, and identification of strain-transcendent rif and stevor genes.