Michael Bastiani

Loss of glial lazarillo, a homolog of apolipoprotein D, reduces lifespan and stress resistance in Drosophila.

Targeted mutagenesis by homologous recombination in D. melanogaster.

The IgLON protein Lachesin is required for the blood-brain barrier in Drosophila.

Axons break in animals lacking beta-spectrin.

Overexpression of PPK-1, the Caenorhabditis elegans Type I PIP kinase, inhibits growth cone collapse in the developing nervous system and causes axonal degeneration in adults.

Apolipoprotein D is involved in the mechanisms regulating protection from oxidative stress.

Molecular interactions of the neuronal GPI-anchored lipocalin Lazarillo.

Axon regeneration requires a conserved MAP kinase pathway.

Targeted gene deletions in C. elegans using transposon excision.

Axon regeneration requires coordinate activation of p38 and JNK MAPK pathways.

Cell architecture: surrounding muscle cells shape gland cell morphology in the Caenorhabditis elegans pharynx.