Juha Voipio

Postnatal development of rat hippocampal gamma rhythm in vivo.

Spontaneous epileptiform activity mediated by GABA(A) receptors and gap junctions in the rat hippocampal slice following long-term exposure to GABA(B) antagonists.

BDNF-induced TrkB activation down-regulates the K+-Cl- cotransporter KCC2 and impairs neuronal Cl- extrusion.

Millivolt-scale DC shifts in the human scalp EEG: evidence for a nonneuronal generator.

Cation-chloride co-transporters in neuronal communication, development and trauma.

Post-traumatic hyperexcitability is not caused by impaired buffering of extracellular potassium.

Vagal nerve stimulation induces intermittent hypocapnia.

Carbonic anhydrase isoform VII acts as a molecular switch in the development of synchronous gamma-frequency firing of hippocampal CA1 pyramidal cells.

Nonneuronal origin of CO2-related DC EEG shifts: an in vivo study in the cat.

Mechanism of activity-dependent downregulation of the neuron-specific K-Cl cotransporter KCC2.

GABA uptake via GABA transporter-1 modulates GABAergic transmission in the immature hippocampus.

Two developmental switches in GABAergic signalling: the K+-Cl- cotransporter KCC2 and carbonic anhydrase CAVII.

Full-band EEG (FbEEG): an emerging standard in electroencephalography.

Carbonic anhydrase inhibitors. Inhibition of the human cytosolic isozyme VII with aromatic and heterocyclic sulfonamides.

Distinct properties of functional KCC2 expression in immature mouse hippocampal neurons in culture and in acute slices.

Depolarizing GABA acts on intrinsically bursting pyramidal neurons to drive giant depolarizing potentials in the immature hippocampus.

Full-band EEG (fbEEG): a new standard for clinical electroencephalography.

Slow endogenous activity transients and developmental expression of K+-Cl- cotransporter 2 in the immature human cortex.

The cation-chloride cotransporter NKCC1 promotes sharp waves in the neonatal rat hippocampus.

Intrinsic bursting of immature CA3 pyramidal neurons and consequent giant depolarizing potentials are driven by a persistent Na+ current and terminated by a slow Ca2+ -activated K+ current.

Experimental febrile seizures are precipitated by a hyperthermia-induced respiratory alkalosis.

GAT-1 acts to limit a tonic GABA(A) current in rat CA3 pyramidal neurons at birth.

Mice with targeted Slc4a10 gene disruption have small brain ventricles and show reduced neuronal excitability.

GABAergic depolarization of the axon initial segment in cortical principal neurons is caused by the Na-K-2Cl cotransporter NKCC1.

Very slow EEG fluctuations predict the dynamics of stimulus detection and oscillation amplitudes in humans.

Compensatory enhancement of intrinsic spiking upon NKCC1 disruption in neonatal hippocampus.

The K+-Cl cotransporter KCC2 promotes GABAergic excitation in the mature rat hippocampus.

Brain alkalosis causes birth asphyxia seizures, suggesting therapeutic strategy.