Robert Foronjy

Emphysematous changes are caused by degradation of type III collagen in transgenic mice expressing MMP-1.

Progressive adult-onset emphysema in transgenic mice expressing human MMP-1 in the lung.

Mechanical unloading during left ventricular assist device support increases left ventricular collagen cross-linking and myocardial stiffness.

Structural emphysema does not correlate with lung compliance: lessons from the mouse smoking model.

Superoxide dismutase expression attenuates cigarette smoke- or elastase-generated emphysema in mice.

Feasibility of retinoids for the treatment of emphysema study.

The impact of angiotensin-converting enzyme inhibitor therapy on the extracellular collagen matrix during left ventricular assist device support in patients with end-stage heart failure.

The pharmokinetic limitations of antioxidant treatment for COPD.

Nonmalignant respiratory effects of chronic arsenic exposure from drinking water among never-smokers in Bangladesh.

Transgenic expression of matrix metalloproteinase-9 causes adult-onset emphysema in mice associated with the loss of alveolar elastin.

Transgenic expression of matrix metalloproteinase-1 inhibits myocardial fibrosis and prevents the transition to heart failure in a pressure overload mouse model.

Particulate matter in cigarette smoke alters iron homeostasis to produce a biological effect.

One-step isolation of plasma membrane proteins using magnetic beads with immobilized concanavalin A.

Role for cathepsin K in emphysema in smoke-exposed guinea pigs.

Eosinophil and T cell markers predict functional decline in COPD patients.

The divergent roles of secreted frizzled related protein-1 (SFRP1) in lung morphogenesis and emphysema.

Protein phosphatase 2A regulates innate immune and proteolytic responses to cigarette smoke exposure in the lung.

Antioxidants of therapeutic relevance in COPD from the neotropical blueberry Anthopterus wardii.

Edible Myrciaria vexator fruits: bioactive phenolics for potential COPD therapy.

Functional characterization of the matrix metalloproteinase-1 cigarette smoke-responsive region and association with the lung health study.

A comparison of the inflammatory and proteolytic effects of dung biomass and cigarette smoke exposure in the lung.

Increased matrix metalloproteinase (MMPs) levels do not predict disease severity or progression in emphysema.

The GPx1-PTP1B-PP2A Axis: A Key Determinant of Airway Inflammation and Alveolar Destruction.

The Potential for Resident Lung Mesenchymal Stem Cells to Promote Functional Tissue Regeneration: Understanding Microenvironmental Cues.