Travis L. Knuckles

Gasoline exhaust emissions induce vascular remodeling pathways involved in atherosclerosis.

Fine oil combustion particle bioavailable constituents induce molecular profiles of oxidative stress, altered function, and cellular injury in cardiomyocytes.

Diesel exhaust exposure enhances venoconstriction via uncoupling of eNOS.

Vehicular emissions induce vascular MMP-9 expression and activity associated with endothelin-1-mediated pathways.

Impairment of coronary endothelial cell ET(B) receptor function after short-term inhalation exposure to whole diesel emissions.

Inhaled diesel emissions alter atherosclerotic plaque composition in ApoE(-/-) mice.

A comparison of vascular effects from complex and individual air pollutants indicates a role for monoxide gases and volatile hydrocarbons.

Formation of vascular S-nitrosothiols and plasma nitrates/nitrites following inhalation of diesel emissions.

Nanoparticle inhalation alters systemic arteriolar vasoreactivity through sympathetic and cyclooxygenase-mediated pathways.

Xenobiotic particle exposure and microvascular endpoints: a call to arms.

Air pollution particulate matter collected from an appalachian mountaintop mining site induces microvascular dysfunction.