Sohail Z. Husain

Improvement of hepatopulmonary syndrome after transjugular intrahepatic portasystemic shunting: case report and review of literature.

The ryanodine receptor mediates early zymogen activation in pancreatitis.

Cyclic AMP-dependent protein kinase and Epac mediate cyclic AMP responses in pancreatic acini.

Caerulein-induced intracellular pancreatic zymogen activation is dependent on calcineurin.

Microvillus inclusion disease associated with coarctation of the aorta and bicuspid aortic valve.

Cyclic AMP accelerates calcium waves in pancreatic acinar cells.

Recurrent acute pancreatitis associated with propionic acidemia.

Recurrent acute pancreatitis in a child with primary hyperparathyroidism.

Characterisation of a fatty acid and retinol binding protein orthologue from the hookworm Ancylostoma ceylanicum.

Dantrolene mitigates caerulein-induced pancreatitis in vivo in mice.

A comparison of presentation and management trends in acute pancreatitis between infants/toddlers and older children.

Protease activation during in vivo pancreatitis is dependent on calcineurin activation.

Molecular and cellular mechanisms of pancreatic injury.

Low extracellular pH induces damage in the pancreatic acinar cell by enhancing calcium signaling.

What have we learned about acute pancreatitis in children?

Ethanol enhances carbachol-induced protease activation and accelerates Ca2+ waves in isolated rat pancreatic acini.

Jejunal adaptation in a prepubertal boy after total ileal resection and jejunostomy placement: a four-year follow-up.

Novel characterization of drug-associated pancreatitis in children.

Two-day bowel preparation with polyethylene glycol 3350 and bisacodyl: a new, safe, and effective regimen for colonoscopy in children.

Risk factors associated with biliary pancreatitis in children.

Pharmacological and genetic inhibition of calcineurin protects against carbachol-induced pathological zymogen activation and acinar cell injury.

Definitions of pediatric pancreatitis and survey of present clinical practices.

Ryanodine receptors contribute to bile acid-induced pathological calcium signaling and pancreatitis in mice.

The association of primary hyperparathyroidism with pancreatitis.

Acute necrotizing pancreatitis in children.

Bile acids induce pancreatic acinar cell injury and pancreatitis by activating calcineurin.

IP3 receptor type 2 deficiency is associated with a secretory defect in the pancreatic acinar cell and an accumulation of zymogen granules.

Pancreatic acinar cell NF-κB activation due to bile acid exposure is dependent on calcineurin.

Src Dependent Pancreatic Acinar Injury Can Be Initiated Independent of an Increase in Cytosolic Calcium.