Stephanie W. Watts

Upregulation of arterial serotonin 1B and 2B receptors in deoxycorticosterone acetate-salt hypertension.

Serotonin-induced contraction in mesenteric resistance arteries: signaling and changes in deoxycorticosterone acetate-salt hypertension.

Endothelin receptor function in mesenteric veins from deoxycorticosterone acetate salt-hypertensive rats.

Endothelin-1-induced venous contraction is maintained in DOCA-salt hypertension; studies with receptor agonists.

Phosphoinositide 3-kinase mediates enhanced spontaneous and agonist-induced contraction in aorta of deoxycorticosterone acetate-salt hypertensive rats.

5-Hydroxytryptamine(2B) receptor function is enhanced in the N(omega)-nitro-L-arginine hypertensive rat.

Endothelin-1 increases vascular superoxide via endothelin(A)-NADPH oxidase pathway in low-renin hypertension.

PI3-kinase-induced hyperreactivity in DOCA-salt hypertension is independent of GSK-3 activity.

NADPH oxidase-derived superoxide augments endothelin-1-induced venoconstriction in mineralocorticoid hypertension.

Arterial expression of 5-HT2B and 5-HT1B receptors during development of DOCA-salt hypertension.

Low [Mg2+]e enhances arterial spontaneous tone via phosphatidylinositol 3-kinase in DOCA-salt hypertension.

Characterization of the contractile 5-hydroxytryptamine receptor in the renal artery of the normotensive rat.

The fenfluramine metabolite (+)-norfenfluramine is vasoactive.

Rho/Rho kinase and phosphoinositide 3-kinase are parallel pathways in the development of spontaneous arterial tone in deoxycorticosterone acetate-salt hypertension.

PI3-kinase upregulation and involvement in spontaneous tone in arteries from DOCA-salt rats: is p110delta the culprit?

Increased O2*- production and upregulation of ETB receptors by sympathetic neurons in DOCA-salt hypertensive rats.

Arteries and veins desensitize differently to endothelin.

Increased vascular sensitivity and connexin43 expression after sympathetic denervation.

C3 or not C3: that is the question.

The serotonin transporter is present and functional in peripheral arterial smooth muscle.

Effects of PPAR-gamma ligands on vascular smooth muscle marker expression in hypertensive and normal arteries.

Endothelin in the splanchnic vascular bed of DOCA-salt hypertensive rats.

Preserving and promoting our discipline: pharmacology students speak out.

5-HT in systemic hypertension: foe, friend or fantasy?

(+)-Norfenfluramine-induced arterial contraction is not dependent on endogenous 5-hydroxytryptamine or 5-hydroxytryptamine transporter.

Endothelin-1-induced contraction in veins is independent of hydrogen peroxide.

Upregulated function of phosphatidylinositol-3-kinase in genetically hypertensive rats: a moderator of arterial hypercontractility.

Activation of vascular BK channel by tempol in DOCA-salt hypertensive rats.

Vasoconstriction caused by the ATP synthase subunit-coupling factor 6: a new function for a historical enzyme.

Pleiotropic effects of hydrogen peroxide in arteries and veins from normotensive and hypertensive rats.

Arterial 5-hydroxytryptamine transporter function is impaired in deoxycorticosterone acetate and Nomega-nitro-L-arginine but not spontaneously hypertensive rats.

5-hydroxytryptamine in the cardiovascular system: focus on the serotonin transporter (SERT).

Mechanisms of hypertension induced by nitric oxide (NO) deficiency: focus on venous function.

A new signaling paradigm for serotonin: use of Crk-associated substrate in arterial contraction.

Cyclooxygenase, p38 mitogen-activated protein kinase (MAPK), extracellular signal-regulated kinase MAPK, Rho kinase, and Src mediate hydrogen peroxide-induced contraction of rat thoracic aorta and vena cava.

A comparison of arteries and veins in oxidative stress: producers, destroyers, function, and disease.

Morphological and biochemical characterization of remodeling in aorta and vena cava of DOCA-salt hypertensive rats.

The 5-hydroxytryptamine2A receptor is involved in (+)-norfenfluramine-induced arterial contraction and blood pressure increase in deoxycorticosterone acetate-salt hypertension.

Preferential myosin heavy chain isoform B Expression may contribute to the faster velocity of contraction in veins versus arteries.

Methyl-beta-cyclodextrin prevents angiotensin II-induced tachyphylactic contractile responses in rat aorta.

Serotonin (5-HT) in veins: not all in vain.

The cytochrome p450 inhibitor ketoconazole potentiates 5-hydroxytryptamine-induced contraction in rat aorta.

Big ET-1 processing into vasoactive peptides in arteries and veins.

Vascular reactivity, 5-HT uptake, and blood pressure in the serotonin transporter knockout rat.

Role of redox signaling and poly (adenosine diphosphate-ribose) polymerase activation in vascular smooth muscle cell growth inhibition by nitric oxide and peroxynitrite.

5-Hydroxytryptamine lowers blood pressure in normotensive and hypertensive rats.

A serotonergic system in veins: serotonin transporter-independent uptake.

Pharmacological endothelin receptor interaction does not occur in veins from ET(B) receptor deficient rats.

Lack of the serotonin transporter does not prevent mineralocorticoid hypertension in rat and mouse.

A comparison of reactive oxygen species metabolism in the rat aorta and vena cava: focus on xanthine oxidase.

The love of a lifetime: 5-HT in the cardiovascular system.

The beginning of a fantastic, unanswered question: is 5-HT involved in systemic hypertension?

Body distribution of infused serotonin in rats.

Endothelin ET(B) receptors in arteries and veins: multiple actions in the vein.

Serotonylation of vascular proteins important to contraction.

Differential expression of pancreatitis-associated protein and thrombospondins in arterial versus venous tissues.

Endothelin receptors: what's new and what do we need to know?

Uric acid does not affect the acetylcholine-induced relaxation of aorta from normotensive and deoxycorticosterone acetate-salt hypertensive rats.

Whole grape intake impacts cardiac peroxisome proliferator-activated receptor and nuclear factor kappaB activity and cytokine expression in rats with diastolic dysfunction.

Serotonin receptors in rat jugular vein: presence and involvement in the contraction.

5-hydroxtryptamine receptors in systemic hypertension: an arterial focus.

The interdependence of endothelin-1 and calcium: a review.

Allopurinol does not decrease blood pressure or prevent the development of hypertension in the deoxycorticosterone acetate-salt rat model.

Serotonin transporter interacts with the PDGFβ receptor in PDGF-BB-induced signaling and mitogenesis in pulmonary artery smooth muscle cells.

Comparison of the function of the serotonin transporter in the vasculature of male and female rats.

Characterization of vabicaserin (SCA-136), a selective 5-hydroxytryptamine 2C receptor agonist.

Modification of proteins by norepinephrine is important for vascular contraction.

Lack of the serotonin transporter (SERT) reduces the ability of 5-hydroxytryptamine to lower blood pressure.

Indoleamine 2,3-diooxygenase in periaortic fat: mechanisms of inhibition of contraction.

Drug Delivery: Enabling Technology for Drug Discovery and Development. iPRECIO Micro Infusion Pump: Programmable, Refillable, and Implantable.

Vena Cava and Aortic Smooth Muscle Cells Express Transglutaminases 1 and 4 in addition to Transglutaminase 2.

Smooth muscle pharmacology in the isolated virgin and pregnant rat uterus and cervix.

Serotonin and blood pressure regulation.

5-hydroxytryptamine (5-HT) reduces total peripheral resistance during chronic infusion: direct arterial mesenteric relaxation is not involved.

Reverse-mode Na+/Ca2+ exchange is an important mediator of venous contraction.

One-Month Serotonin Infusion Results in a Prolonged Fall in Blood Pressure in the Deoxycorticosterone Acetate (DOCA) Salt Hypertensive Rat.

Long-Term Inhibition of Xanthine Oxidase by Febuxostat Does Not Decrease Blood Pressure in Deoxycorticosterone Acetate (DOCA)-Salt Hypertensive Rats.

Chemerin connects fat to arterial contraction.

Divergent signaling mechanisms for venous versus arterial contraction as revealed by endothelin-1.

The persistence of active smooth muscle in the female rat cervix through pregnancy.

Serotonin and sensory nerves: meeting in the cardiovascular system.

Elimination of vitamin D receptor in vascular endothelial cells alters vascular function.

Tissue transglutaminase promotes serotonin-induced AKT signaling and mitogenesis in pulmonary vascular smooth muscle cells.

Transglutaminase activity is decreased in large arteries from hypertensive rats compared with normotensive controls.

5-HT is a potent relaxant in rat superior mesenteric veins.

5-Hydroxytryptamine does not reduce sympathetic nerve activity or neuroeffector function in the splanchnic circulation.

Trash Talk by Fat: Chemerin as a Reactive Oxygen Species Provocateur in the Vasculature.

Chemerin: A comprehensive review elucidating the need for cardiovascular research.

An immunohistochemical analysis of SERT in the blood-brain barrier of the male rat brain.

Organic cation transporter 3 contributes to norepinephrine uptake into perivascular adipose tissue.

Introducing a checklist for manuscript submission to Pharmacological Research.

Pharmacological research and precision cancer medicine: A call for manuscripts.

Serial Measurements of Splanchnic Vein Diameters in Rats Using High-Frequency Ultrasound.

The adipokine chemerin amplifies electrical field-stimulated contraction in the isolated rat superior mesenteric artery.

Oh, the places you'll go! My many colored serotonin (apologies to Dr. Seuss).

The distribution and adipogenic potential of perivascular adipose tissue adipocyte progenitors is dependent on sexual dimorphism and vessel location.

New actions of an old friend: perivascular adipose tissue's adrenergic mechanisms.

Chemerin-induced arterial contraction is Gi- and calcium-dependent.

3T3-L1 cells and perivascular adipocytes are not equivalent in amine transporter expression.

Regulator of G Protein Signaling 6 Protects the Heart from Ischemic Injury.

Perivascular Adipose Tissue's Impact on Norepinephrine-Induced Contraction of Mesenteric Resistance Arteries.

5-HT causes splanchnic venodilation.