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Department of Neuropathology,
Graduate School of Medicine,
Department of Neuropathology, Graduate School of Medicine
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[Involvement of ouabain (digitalis-like factor) in the pathogenesis of hypertension and hypertensive target-organ damage].
Nihon rinsho. Japanese journal of clinical medicine Mar, 2004 | Pubmed ID: 15171364
The low density lipoprotein receptor-related protein 1 mediates uptake of amyloid beta peptides in an in vitro model of the blood-brain barrier cells.
The Journal of biological chemistry Dec, 2008 | Pubmed ID: 18940800
Abeta immunotherapy: intracerebral sequestration of Abeta by an anti-Abeta monoclonal antibody 266 with high affinity to soluble Abeta.
The Journal of neuroscience : the official journal of the Society for Neuroscience Sep, 2009 | Pubmed ID: 19741145
Halichlorine is a novel L-type Ca(2+) channel inhibitor isolated from the marine sponge Halichondria okadai Kadota.
European journal of pharmacology Feb, 2010 | Pubmed ID: 19919831
Halichlorine reduces monocyte adhesion to endothelium through the suppression of nuclear factor-kappaB activation.
Journal of pharmacological sciences , 2010 | Pubmed ID: 20562517
Effects of CD2-associated protein deficiency on amyloid-β in neuroblastoma cells and in an APP transgenic mouse model.
Molecular neurodegeneration Mar, 2015 | Pubmed ID: 25887956
Analysis of in vivo turnover of tau in a mouse model of tauopathy.
Molecular neurodegeneration Oct, 2015 | Pubmed ID: 26502977
In Vivo Microdialysis of Brain Interstitial Fluid for the Determination of Extracellular Tau Levels.
Methods in molecular biology (Clifton, N.J.) , 2017 | Pubmed ID: 27975257
ApoE4 markedly exacerbates tau-mediated neurodegeneration in a mouse model of tauopathy.
Nature 09, 2017 | Pubmed ID: 28959956
Extracellular Tau and Its Potential Role in the Propagation of Tau Pathology.
Frontiers in neuroscience , 2017 | Pubmed ID: 29238289
Extracellular α-synuclein levels are regulated by neuronal activity.
Molecular neurodegeneration 02, 2018 | Pubmed ID: 29467003
University of Tokyo
Kaoru Yamada1
1Department of Neuropathology, Graduate School of Medicine, University of Tokyo
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