Maud Frieden

Tissue-specific expression of human lipoprotein lipase in the vascular system affects vascular reactivity in transgenic mice.

Subplasmalemmal endoplasmic reticulum controls K(Ca) channel activity upon stimulation with a moderate histamine concentration in a human umbilical vein endothelial cell line.

Calreticulin differentially modulates calcium uptake and release in the endoplasmic reticulum and mitochondria.

Mitochondria efficiently buffer subplasmalemmal Ca2+ elevation during agonist stimulation.

Measurements of the free luminal ER Ca(2+) concentration with targeted "cameleon" fluorescent proteins.

Sustained Ca2+ transfer across mitochondria is Essential for mitochondrial Ca2+ buffering, sore-operated Ca2+ entry, and Ca2+ store refilling.

Ca(2+) homeostasis during mitochondrial fragmentation and perinuclear clustering induced by hFis1.

The role of mitochondria for Ca2+ refilling of the endoplasmic reticulum.

Twenty years of calcium imaging: cell physiology to dye for.

Subplasmalemmal mitochondria modulate the activity of plasma membrane Ca2+-ATPases.

STIM1 knockdown reveals that store-operated Ca2+ channels located close to sarco/endoplasmic Ca2+ ATPases (SERCA) pumps silently refill the endoplasmic reticulum.

Mitochondria and Ca(2+) signaling: old guests, new functions.

Regulation of plasma membrane calcium fluxes by mitochondria.

Dysfunction of mitochondria Ca2+ uptake in cystic fibrosis airway epithelial cells.

SLP-2 negatively modulates mitochondrial sodium-calcium exchange.

Electrophysiological characterization of store-operated and agonist-induced Ca2+ entry pathways in endothelial cells.

Thapsigargin activates Ca²+ entry both by store-dependent, STIM1/Orai1-mediated, and store-independent, TRPC3/PLC/PKC-mediated pathways in human endothelial cells.

Remodelling of the endoplasmic reticulum during store-operated calcium entry.

Local cytosolic Ca2+ elevations are required for stromal interaction molecule 1 (STIM1) de-oligomerization and termination of store-operated Ca2+ entry.

Transient receptor potential canonical channels are required for in vitro endothelial tube formation.

Activation of transient receptor potential canonical 3 (TRPC3)-mediated Ca2+ entry by A1 adenosine receptor in cardiomyocytes disturbs atrioventricular conduction.

During post-natal human myogenesis, normal myotube size requires TRPC1- and TRPC4-mediated Ca²⁺ entry.

Feature Article: mTOR complex 2-Akt signaling at mitochondria-associated endoplasmic reticulum membranes (MAM) regulates mitochondrial physiology.

Nicotine mediates oxidative stress and apoptosis through cross talk between NOX1 and Bcl-2 in lung epithelial cells.

Inositol 1,4,5 trisphosphate receptor 1 is a key player of human myoblast differentiation.

Airway Epithelial Cell Integrity Protects from Cytotoxicity of Pseudomonas aeruginosa Quorum-Sensing Signals.

SERCA and PMCA pumps contribute to the deregulation of Ca2+ homeostasis in human CF epithelial cells.

STIM1L traps and gates Orai1 channels without remodeling the cortical ER.

TRPC1 and TRPC4 channels functionally interact with STIM1L to promote myogenesis and maintain fast repetitive Ca(2+) release in human myotubes.

Calumenin contributes to ER-Ca(2+) homeostasis in bronchial epithelial cells expressing WT and F508del mutated CFTR and to F508del-CFTR retention.