Sanda Predescu

Nitric oxide stimulates macrophage inflammatory protein-2 expression in sepsis.

Transport of nitrated albumin across continuous vascular endothelium.

Intersectin regulates fission and internalization of caveolae in endothelial cells.

G alpha12 interaction with alphaSNAP induces VE-cadherin localization at endothelial junctions and regulates barrier function.

Constitutive eNOS-derived nitric oxide is a determinant of endothelial junctional integrity.

Cholesterol-dependent syntaxin-4 and SNAP-23 clustering regulates caveolar fusion with the endothelial plasma membrane.

siRNA-induced caveolin-1 knockdown in mice increases lung vascular permeability via the junctional pathway.

A novel lysophospholipid- and pH-sensitive receptor, GPR4, in brain endothelial cells regulates monocyte transmigration.

Intersectin-1s regulates the mitochondrial apoptotic pathway in endothelial cells.

Molecular determinants of endothelial transcytosis and their role in endothelial permeability.

Tiam1 and Rac1 are required for platelet-activating factor-induced endothelial junctional disassembly and increase in vascular permeability.

Intersectin-2L regulates caveola endocytosis secondary to Cdc42-mediated actin polymerization.

Regulation of dynamin-2 assembly-disassembly and function through the SH3A domain of intersectin-1s.

Stress chaperone GRP-78 functions in mineralized matrix formation.

Pro-inflammatory endothelial cell dysfunction is associated with intersectin-1s down-regulation.

Impaired caveolae function and upregulation of alternative endocytic pathways induced by experimental modulation of intersectin-1s expression in mouse lung endothelium.

In vivo knockdown of intersectin-1s alters endothelial cell phenotype and causes microvascular remodeling in the mouse lungs.

Conditional deletion of FAK in mice endothelium disrupts lung vascular barrier function due to destabilization of RhoA and Rac1 activities.