Gary M. Mawe

Nerves and Hormones Interact to Control Gallbladder Function.

Ca(2+) sparks and BK currents in gallbladder myocytes: role in CCK-induced response.

Opioid agonists inhibit excitatory neurotransmission in ganglia and at the neuromuscular junction in Guinea pig gallbladder.

Effects of PGE2 in guinea pig colonic myenteric ganglia.

ERG K+ channels modulate the electrical and contractile activities of gallbladder smooth muscle.

Neuroimmune and epithelial interactions in intestinal inflammation.

Enhanced excitability of myenteric AH neurones in the inflamed guinea-pig distal colon.

Effects of bioactive agents on biliary motor function.

Serotonin availability is increased in mucosa of guinea pigs with TNBS-induced colitis.

Distribution and chemical coding of cocaine- and amphetamine-regulated transcript peptide (CART)-immunoreactive neurons in the guinea pig bowel.

Enterochromaffin cells and 5-HT signaling in the pathophysiology of disorders of gastrointestinal function.

Cyclooxygenase-2 contributes to dysmotility and enhanced excitability of myenteric AH neurones in the inflamed guinea pig distal colon.

Molecular defects in mucosal serotonin content and decreased serotonin reuptake transporter in ulcerative colitis and irritable bowel syndrome.

Inflammatory neuropathies of the enteric nervous system.

Enteroendocrine cells and 5-HT availability are altered in mucosa of guinea pigs with TNBS ileitis.

Innervation of the extrahepatic biliary tract.

Characterization of intracellular Ca(2+) stores in gallbladder smooth muscle.

Cyclic AMP-mediated inhibition of gallbladder contractility: role of K+ channel activation and Ca2+ signaling.

Disruption of the filamentous actin cytoskeleton is necessary for the activation of capacitative calcium entry in naive smooth muscle cells.

Synaptic facilitation and enhanced neuronal excitability in the submucosal plexus during experimental colitis in guinea-pig.

Identification of a spontaneously active, Na+-permeable channel in guinea pig gallbladder smooth muscle.

Spontaneous electrical rhythmicity and the role of the sarcoplasmic reticulum in the excitability of guinea pig gallbladder smooth muscle cells.

Effects of gastrointestinal inflammation on enteroendocrine cells and enteric neural reflex circuits.

Calcium waves in intact guinea pig gallbladder smooth muscle cells.

Ileitis alters neuronal and enteroendocrine signalling in guinea pig distal colon.

Persistent alterations to enteric neural signaling in the guinea pig colon following the resolution of colitis.

IFN-gamma and TNF-alpha decrease serotonin transporter function and expression in Caco2 cells.

Serotonin and its role in colonic function and in gastrointestinal disorders.

Morphological and physiological evidence for interstitial cell of Cajal-like cells in the guinea pig gallbladder.

Synaptic plasticity in myenteric neurons of the guinea-pig distal colon: presynaptic mechanisms of inflammation-induced synaptic facilitation.

Role of mitochondria in spontaneous rhythmic activity and intracellular calcium waves in the guinea pig gallbladder smooth muscle.

ATP induces guinea pig gallbladder smooth muscle excitability via the P2Y4 receptor and COX-1 activity.

Serotonin signaling in diverticular disease.

Alterations to enteric neural signaling underlie secretory abnormalities of the ileum in experimental colitis in the guinea pig.

The effects of daikenchuto (DKT) on propulsive motility in the colon.

Interstitial cells of Cajal in the gut: what makes them tick?

Mucosal serotonin signaling is altered in chronic constipation but not in opiate-induced constipation.

Purinergic neuromuscular transmission is selectively attenuated in ulcerated regions of inflamed guinea pig distal colon.

Serotonin signaling is altered in irritable bowel syndrome with diarrhea but not in functional dyspepsia in pediatric age patients.

Hydrophobic bile salts inhibit gallbladder smooth muscle function via stimulation of GPBAR1 receptors and activation of KATP channels.

Neurogastroenterology in 2011: Emerging concepts in neurogastroenterology and motility.

Activation of Colonic Mucosal 5-HT(4) Receptors Accelerates Propulsive Motility and Inhibits Visceral Hypersensitivity.