Simon Cuhlmann

Increased endothelial mitogen-activated protein kinase phosphatase-1 expression suppresses proinflammatory activation at sites that are resistant to atherosclerosis.

Activation of Nrf2 in endothelial cells protects arteries from exhibiting a proinflammatory state.

c-Jun N-terminal kinase primes endothelial cells at atheroprone sites for apoptosis.

Role of nuclear factor kappaB in cardiovascular health and disease.

Disturbed blood flow induces RelA expression via c-Jun N-terminal kinase 1: a novel mode of NF-κB regulation that promotes arterial inflammation.