Kathryn N. Farrow

Structural and functional analysis of the differential effects of c-Jun and v-Jun on prolactin gene expression.

The diseases treated with ECMO: focus on PPHN.

eNOS function is developmentally regulated: uncoupling of eNOS occurs postnatally.

Hyperoxia increases phosphodiesterase 5 expression and activity in ovine fetal pulmonary artery smooth muscle cells.

Superoxide dismutase restores eNOS expression and function in resistance pulmonary arteries from neonatal lambs with persistent pulmonary hypertension.

Milrinone enhances relaxation to prostacyclin and iloprost in pulmonary arteries isolated from lambs with persistent pulmonary hypertension of the newborn.

Regulation of phosphodiesterase 3 in the pulmonary arteries during the perinatal period in sheep.

Hypoxia increases ROS signaling and cytosolic Ca(2+) in pulmonary artery smooth muscle cells of mouse lungs slices.

SOD and inhaled nitric oxide normalize phosphodiesterase 5 expression and activity in neonatal lambs with persistent pulmonary hypertension.

Mitochondrial oxidant stress increases PDE5 activity in persistent pulmonary hypertension of the newborn.

Phosphodiesterases: emerging therapeutic targets for neonatal pulmonary hypertension.

Apocynin improves oxygenation and increases eNOS in persistent pulmonary hypertension of the newborn.

Hydrocortisone normalizes oxygenation and cGMP regulation in lambs with persistent pulmonary hypertension of the newborn.

Brief hyperoxia increases mitochondrial oxidation and increases phosphodiesterase 5 activity in fetal pulmonary artery smooth muscle cells.

Cyclic stretch induces inducible nitric oxide synthase and soluble guanylate cyclase in pulmonary artery smooth muscle cells.

Postnatal Weight Gain in Preterm Infants with Severe Bronchopulmonary Dysplasia.

Disrupted Pulmonary Artery cGMP Signaling in Mice with Hyperoxia-Induced Pulmonary Hypertension.