Gunter Leuckx

Ectopic expression of E2F1 stimulates beta-cell proliferation and function.

Adult duct-lining cells can reprogram into β-like cells able to counter repeated cycles of toxin-induced diabetes.

Mouse beta cell proliferation is inhibited by thymidine analogue labelling.

The inactivation of Arx in pancreatic α-cells triggers their neogenesis and conversion into functional β-like cells.

Reversal of hyperglycemia in diabetic mice by a marginal islet mass together with human blood outgrowth endothelial cells is independent of the delivery technique and blood clot-induced processes.

Transient cytokine treatment induces acinar cell reprogramming and regenerates functional beta cell mass in diabetic mice.

IL-6-dependent proliferation of alpha cells in mice with partial pancreatic-duct ligation.

Partial duct ligation: β-cell proliferation and beyond.

Macrophage dynamics are regulated by local macrophage proliferation and monocyte recruitment in injured pancreas.

Concise Review: Macrophages: Versatile Gatekeepers During Pancreatic β-Cell Development, Injury, and Regeneration.

Estrogen Receptor α Regulates Beta Cell Formation During Pancreas Development and Following Injury.