Soledad Galli

Mitochondrial nitric oxide synthase drives redox signals for proliferation and quiescence in rat liver development.

Decreased mitochondrial nitric oxide synthase activity and hydrogen peroxide relate persistent tumoral proliferation to embryonic behavior.

Cell H2O2 steady-state concentration and mitochondrial nitric oxide.

Hypothyroid phenotype is contributed by mitochondrial complex I inactivation due to translocated neuronal nitric-oxide synthase.

A mitochondrial kinase complex is essential to mediate an ERK1/2-dependent phosphorylation of a key regulatory protein in steroid biosynthesis.

Tumor cell phenotype is sustained by selective MAPK oxidation in mitochondria.

Akt1 intramitochondrial cycling is a crucial step in the redox modulation of cell cycle progression.

A new paradigm for MAPK: structural interactions of hERK1 with mitochondria in HeLa cells.

New algorithm to determine true colocalization in combination with image restoration and time-lapse confocal microscopy to MAP kinases in mitochondria.

Structural model for p75(NTR)-TrkA intracellular domain interaction: a combined FRET and bioinformatics study.

Dysfunction of Wnt signaling and synaptic disassembly in neurodegenerative diseases.

Deficient Wnt signalling triggers striatal synaptic degeneration and impaired motor behaviour in adult mice.

Reversal of Synapse Degeneration by Restoring Wnt Signaling in the Adult Hippocampus.