Theresa Pizarro

Identification of a quantitative trait locus for ileitis in a spontaneous mouse model of Crohn's disease: SAMP1/YitFc.

RNA binding properties of the AU-rich element-binding recombinant Nup475/TIS11/tristetraprolin protein.

Genetic dissection of the cellular pathways and signaling mechanisms in modeled tumor necrosis factor-induced Crohn's-like inflammatory bowel disease.

Emergence of perianal fistulizing disease in the SAMP1/YitFc mouse, a spontaneous model of chronic ileitis.

Mouse models for the study of Crohn's disease.

TNF-alpha neutralization ameliorates the severity of murine Crohn's-like ileitis by abrogation of intestinal epithelial cell apoptosis.

Expression, localization, and functional activity of TL1A, a novel Th1-polarizing cytokine in inflammatory bowel disease.

Expanded B cell population blocks regulatory T cells and exacerbates ileitis in a murine model of Crohn disease.

Commentary: the role of the IL-18 system and other members of the IL-1R/TLR superfamily in innate mucosal immunity and the pathogenesis of inflammatory bowel disease: friend or foe?

Altered epithelial cell lineage allocation and global expansion of the crypt epithelial stem cell population are associated with ileitis in SAMP1/YitFc mice.

Targeting mucosal addressin cellular adhesion molecule (MAdCAM)-1 to noninvasively image experimental Crohn's disease.

The primary defect in experimental ileitis originates from a nonhematopoietic source.

Role of TL1A and its receptor DR3 in two models of chronic murine ileitis.

Cytokine therapy for Crohn's disease: advances in translational research.

Commensal bacteria exacerbate intestinal inflammation but are not essential for the development of murine ileitis.

Does gender affect career satisfaction and advancement in gastroenterology? Results of an AGA institute-sponsored survey.

Cloning IL-1 and the birth of a new era in cytokine biology.

MNK kinases regulate multiple TLR pathways and innate proinflammatory cytokines in macrophages.

Innate and adaptive immune responses related to IBD pathogenesis.

Mechanisms of tight junction dysregulation in the SAMP1/YitFc model of Crohn's disease-like ileitis.

Emerging drugs for the treatment of ulcerative colitis.

Probiotics promote gut health through stimulation of epithelial innate immunity.

Epithelial-derived IL-33 and its receptor ST2 are dysregulated in ulcerative colitis and in experimental Th1/Th2 driven enteritis.

Beta7 integrin deficiency suppresses B cell homing and attenuates chronic ileitis in SAMP1/YitFc mice.

IL-1 family nomenclature.

SAMP1/YitFc mouse strain: a spontaneous model of Crohn's disease-like ileitis.

Spontaneous, immune-mediated gastric inflammation in SAMP1/YitFc mice, a model of Crohn's-like gastritis.

Novel cytokine signaling pathways in inflammatory bowel disease: insight into the dichotomous functions of IL-33 during chronic intestinal inflammation.

New insights into the dichotomous role of innate cytokines in gut homeostasis and inflammation.

Probiotic bacteria regulate intestinal epithelial permeability in experimental ileitis by a TNF-dependent mechanism.

Emerging role of the interleukin (IL)-33/ST2 axis in gut mucosal wound healing and fibrosis.

Challenges in IBD Research: Update on Progress and Prioritization of the CCFA's Research Agenda.

Associations between genetic polymorphisms in IL-33, IL1R1 and risk for inflammatory bowel disease.

The role of IL-33 in gut mucosal inflammation.

Opposing Functions of Classic and Novel IL-1 Family Members in Gut Health and Disease.

Intestinal-Specific TNFα Overexpression Induces Crohn's-Like Ileitis in Mice.

Central Role of the Gut Epithelial Barrier in the Pathogenesis of Chronic Intestinal Inflammation: Lessons Learned from Animal Models and Human Genetics.

Dysregulated NOD2 predisposes SAMP1/YitFc mice to chronic intestinal inflammation.

Regulatory cell populations in the intestinal mucosa.