Rachid Mazroui

Trapping of messenger RNA by Fragile X Mental Retardation protein into cytoplasmic granules induces translation repression.

Fragile X Mental Retardation protein determinants required for its association with polyribosomal mRNPs.

The fragile X mental retardation protein has nucleic acid chaperone properties.

Biochemical evidence for the association of fragile X mental retardation protein with brain polyribosomal ribonucleoparticles.

The RNA-binding protein fragile X-related 1 regulates somite formation in Xenopus laevis.

NF-kappa B-mediated MyoD decay during muscle wasting requires nitric oxide synthase mRNA stabilization, HuR protein, and nitric oxide release.

Inhibition of ribosome recruitment induces stress granule formation independently of eukaryotic initiation factor 2alpha phosphorylation.

Inhibition of the ubiquitin-proteasome system induces stress granule formation.

The RNA-binding protein HuR promotes cell migration and cell invasion by stabilizing the beta-actin mRNA in a U-rich-element-dependent manner.

Caspase-mediated cleavage of HuR in the cytoplasm contributes to pp32/PHAP-I regulation of apoptosis.

TDRD3, a novel Tudor domain-containing protein, localizes to cytoplasmic stress granules.

Uncoupling stress granule assembly and translation initiation inhibition.

The chemotherapeutic agent bortezomib induces the formation of stress granules.

p21(WAF1/CIP1) upregulation through the stress granule-associated protein CUGBP1 confers resistance to bortezomib-mediated apoptosis.

Turnover of AU-rich-containing mRNAs during stress: a matter of survival.

The translation inhibitor pateamine A prevents cachexia-induced muscle wasting in mice.

Characterization of Fragile X Mental Retardation Protein granules formation and dynamics in Drosophila.

Characterization of fragile X mental retardation protein recruitment and dynamics in Drosophila stress granules.

Inactivation of the mTORC1-eukaryotic translation initiation factor 4E pathway alters stress granule formation.

Gene and MicroRNA transcriptome analysis of Parkinson's related LRRK2 mouse models.