Serge Mostowy

Virulence factors that modulate the cell biology of Listeria infection and the host response.

The evolution of trade-offs: testing predictions on response to selection and environmental variation.

Genomic deletions suggest a phylogeny for the Mycobacterium tuberculosis complex.

Comparative genomics in the fight against tuberculosis: diagnostics, epidemiology, and BCG vaccination.

The in vitro evolution of BCG vaccines.

Genomic interrogation of the dassie bacillus reveals it as a unique RD1 mutant within the Mycobacterium tuberculosis complex.

Genetic characterization of the Guinea-Bissau family of Mycobacterium tuberculosis complex strains.

Genomic characterization of an endemic Mycobacterium tuberculosis strain: evolutionary and epidemiologic implications.

The Mycobacterium tuberculosis complex transcriptome of attenuation.

Genomic analysis distinguishes Mycobacterium africanum.

Extensive genomic polymorphism within Mycobacterium avium.

Point mutations in the DNA- and cNMP-binding domains of the homologue of the cAMP receptor protein (CRP) in Mycobacterium bovis BCG: implications for the inactivation of a global regulator and strain attenuation.

The origin and evolution of Mycobacterium tuberculosis.

Reduced expression of antigenic proteins MPB70 and MPB83 in Mycobacterium bovis BCG strains due to a start codon mutation in sigK.

Revisiting the evolution of Mycobacterium bovis.

Mutations in Mycobacterium tuberculosis Rv0444c, the gene encoding anti-SigK, explain high level expression of MPB70 and MPB83 in Mycobacterium bovis.

Molecular tools for typing and branding the tubercle bacillus.

Molecular genetic analysis of two loci (Ity2 and Ity3) involved in the host response to infection with Salmonella typhimurium using congenic mice and expression profiling.

Autoinducer-2 triggers the oxidative stress response in Mycobacterium avium, leading to biofilm formation.

PhoP: a missing piece in the intricate puzzle of Mycobacterium tuberculosis virulence.

NOD2-deficient mice have impaired resistance to Mycobacterium tuberculosis infection through defective innate and adaptive immunity.

Septins regulate bacterial entry into host cells.

Septin 11 restricts InlB-mediated invasion by Listeria.

Cytoskeleton rearrangements during Listeria infection: clathrin and septins as new players in the game.

From pathogenesis to cell biology and back.

Mycobacterium africanum is not a major cause of human tuberculosis in Cape Town, South Africa.

Entrapment of intracytosolic bacteria by septin cage-like structures.

Autophagy and the cytoskeleton: new links revealed by intracellular pathogens.

A role for septins in the interaction between the Listeria monocytogenes INVASION PROTEIN InlB and the Met receptor.

p62 and NDP52 proteins target intracytosolic Shigella and Listeria to different autophagy pathways.

Septins as key regulators of actin based processes in bacterial infection.

Recruitment of the major vault protein by InlK: a Listeria monocytogenes strategy to avoid autophagy.

Listeria and autophagy escape: involvement of InlK, an internalin-like protein.

Septins: the fourth component of the cytoskeleton.

Bacterial autophagy: restriction or promotion of bacterial replication?

Guidelines for the use and interpretation of assays for monitoring autophagy.

Autophagy and bacterial clearance: a not so clear picture.

Selective autophagy degrades DICER and AGO2 and regulates miRNA activity.

Autophagy selectively regulates miRNA homeostasis.

Mycolactone activation of Wiskott-Aldrich syndrome proteins underpins Buruli ulcer formation.

Septins 2, 7 and 9 and MAP4 colocalize along the axoneme in the primary cilium and control ciliary length.

Species-specific impact of the autophagy machinery on Chikungunya virus infection.

The Zebrafish as a New Model for the In Vivo Study of Shigella flexneri Interaction with Phagocytes and Bacterial Autophagy.

Highlight: the 5th International Workshop on Septin Biology.

Role of endothelial cell septin 7 in the endocytosis of Candida albicans.