Vincent W. Yang

Inducible intestine-specific deletion of Krüppel-like factor 5 is characterized by a regenerative response in adult mouse colon.

Krüppel-like factor 5 is essential for proliferation and survival of mouse intestinal epithelial stem cells.

ML264, A Novel Small-Molecule Compound That Potently Inhibits Growth of Colorectal Cancer.

KLF4 Suppresses Tumor Formation in Genetic and Pharmacological Mouse Models of Colonic Tumorigenesis.

Krüppel-like Factor 4 Modulates Development of BMI1(+) Intestinal Stem Cell-Derived Lineage Following γ-Radiation-Induced Gut Injury in Mice.

Krüppel-like factor 4 (KLF4): What we currently know.

SP and KLF Transcription Factors in Digestive Physiology and Diseases.

Increased Genetic Instability and Accelerated Progression of Colitis-Associated Colorectal Cancer through Intestinal Epithelium-specific Deletion of .

The Novel Small-Molecule SR18662 Efficiently Inhibits the Growth of Colorectal Cancer and .

Krüppel-like Factor 5 Regulates Stemness, Lineage Specification, and Regeneration of Intestinal Epithelial Stem Cells.

Interplay among p21, MUSASHI-1 and Krüppel-like factor 4 in activation of Bmi1-Cre reserve intestinal stem cells after gamma radiation-induced injury.

Aberrant differentiation of intestinal stem cells due to inflammation-induced mitochondrial dysfunction predicts postoperative recurrence of Crohn's disease.

KLF5 protects the intestinal epithelium against Th17 immune response in a murine colitis model.