Kathia Johnson

Role of group II and group III metabotropic glutamate receptors in spinal cord injury.

Group I metabotropic glutamate receptors in spinal cord injury: roles in neuroprotection and the development of chronic central pain.

Locomotor recovery and mechanical hyperalgesia following spinal cord injury depend on age at time of injury in rat.

Effect of age at time of spinal cord injury on behavioral outcomes in rat.

Transcriptional profiling of spinal cord injury-induced central neuropathic pain.

Increases in the activated forms of ERK 1/2, p38 MAPK, and CREB are correlated with the expression of at-level mechanical allodynia following spinal cord injury.

Human fetal neural stem cells grafted into contusion-injured rat spinal cords improve behavior.

Serum albumin improves recovery from spinal cord injury.

Nuclear factor-kappaB decoy amelioration of spinal cord injury-induced inflammation and behavior outcomes.

Detrimental effects of antiapoptotic treatments in spinal cord injury.

Activation of p38 MAP kinase is involved in central neuropathic pain following spinal cord injury.

Regulation of interleukin-1beta by the interleukin-1 receptor antagonist in the glutamate-injured spinal cord: endogenous neuroprotection.

Amiloride improves locomotor recovery after spinal cord injury.

Calcium/calmodulin dependent kinase II contributes to persistent central neuropathic pain following spinal cord injury.

Inflammatory consequences in a rodent model of mild traumatic brain injury.

Reactive oxygen species and lipid peroxidation inhibitors reduce mechanical sensitivity in a chronic neuropathic pain model of spinal cord injury in rats.

Comparison of Mechanical Allodynia and Recovery of Locomotion and Bladder Function by Different Parameters of Low Thoracic Spinal Contusion Injury in Rats.

Persistent Behavioral Deficits in Rats after Parasagittal Fluid Percussion Injury.