Melissa G. Farb

Reduced adipose tissue inflammation represents an intermediate cardiometabolic phenotype in obesity.

Arteriolar function in visceral adipose tissue is impaired in human obesity.

B lymphocytes in human subcutaneous adipose crown-like structures.

The A2b adenosine receptor modulates glucose homeostasis and obesity.

Protein kinase C-β contributes to impaired endothelial insulin signaling in humans with diabetes mellitus.

Cyclooxygenase inhibition improves endothelial vasomotor dysfunction of visceral adipose arterioles in human obesity.

Insulin status and vascular responses to weight loss in obesity.

Antiangiogenic actions of vascular endothelial growth factor-A165b, an inhibitory isoform of vascular endothelial growth factor-A, in human obesity.

Noncanonical Wnt signaling promotes obesity-induced adipose tissue inflammation and metabolic dysfunction independent of adipose tissue expansion.

Visceral adiposopathy: a vascular perspective.

Forkhead box O-1 modulation improves endothelial insulin resistance in human obesity.

Effect of Bariatric Weight Loss on the Adipose Lipolytic Transcriptome in Obese Humans.

Endothelial Dysfunction in Human Diabetes Is Mediated by Wnt5a-JNK Signaling.

Restoration of autophagy in endothelial cells from patients with diabetes mellitus improves nitric oxide signaling.

WNT5A-JNK regulation of vascular insulin resistance in human obesity.

WNT5A regulates adipose tissue angiogenesis via antiangiogenic VEGF-A165b in obese humans.