Mattias Rickhag

Comprehensive regional and temporal gene expression profiling of the rat brain during the first 24 h after experimental stroke identifies dynamic ischemia-induced gene expression patterns, and reveals a biphasic activation of genes in surviving tissue.

Induction of apoptotic cell death by putrescine.

Npas4, a novel helix-loop-helix PAS domain protein, is regulated in response to cerebral ischemia.

Rapid and long-term induction of effector immediate early genes (BDNF, Neuritin and Arc) in peri-infarct cortex and dentate gyrus after ischemic injury in rat brain.

Apolipoprotein D is elevated in oligodendrocytes in the peri-infarct region after experimental stroke: influence of enriched environment.

Enriched environment reduces apolipoprotein E (ApoE) in reactive astrocytes and attenuates inflammation of the peri-infarct tissue after experimental stroke.

The sigma-1 receptor enhances brain plasticity and functional recovery after experimental stroke.

A C-terminal PDZ domain-binding sequence is required for striatal distribution of the dopamine transporter.

PICK1 deficiency impairs secretory vesicle biogenesis and leads to growth retardation and decreased glucose tolerance.

Membrane-permeable C-terminal dopamine transporter peptides attenuate amphetamine-evoked dopamine release.

In vivo amphetamine action is contingent on αCaMKII.

Amphetamine action at the cocaine- and antidepressant-sensitive serotonin transporter is modulated by αCaMKII.

A novel dopamine transporter transgenic mouse line for identification and purification of midbrain dopaminergic neurons reveals midbrain heterogeneity.

Preserved dopaminergic homeostasis and dopamine-related behaviour in hemizygous TH-Cre mice.