Stewart A. Anderson

Directed differentiation and functional maturation of cortical interneurons from human embryonic stem cells.

The protracted maturation of human ESC-derived interneurons.

GABAergic interneuron transplants to study development and treat disease.

The chandelier cell, form and function.

Cortical neurogenesis from pluripotent stem cells: complexity emerging from simplicity.

Cortical parvalbumin GABAergic deficits with α7 nicotinic acetylcholine receptor deletion: implications for schizophrenia.

Spatial distribution of neurons innervated by chandelier cells.

Diversity of cortical interneurons in primates: the role of the dorsal proliferative niche.

Reprogramming patient-derived cells to study the epilepsies.

Duration of culture and sonic hedgehog signaling differentially specify PV versus SST cortical interneuron fates from embryonic stem cells.

Reduction in focal ictal activity following transplantation of MGE interneurons requires expression of the GABAA receptor α4 subunit.

Hopx distinguishes hippocampal from lateral ventricle neural stem cells.

Apical versus Basal Neurogenesis Directs Cortical Interneuron Subclass Fate.

Fate determination of cerebral cortical GABAergic interneurons and their derivation from stem cells.

D-Serine and Serine Racemase Are Associated with PSD-95 and Glutamatergic Synapse Stability.

Differential Mitochondrial Requirements for Radially and Non-radially Migrating Cortical Neurons: Implications for Mitochondrial Disorders.

Stem cell-derived interneuron transplants as a treatment for schizophrenia: preclinical validation in a rodent model.

A viral strategy for targeting and manipulating interneurons across vertebrate species.

Atypical PKC and Notch Inhibition Differentially Modulate Cortical Interneuron Subclass Fate from Embryonic Stem Cells.

Loss of the neurodevelopmental gene Zswim6 alters striatal morphology and motor regulation.

The NANCI-Nkx2.1 gene duplex buffers Nkx2.1 expression to maintain lung development and homeostasis.

Newfound sex differences in axonal structure underlie differential outcomes from in vitro traumatic axonal injury.