Darren Boehning

Circadian rhythms. Carbon monoxide and clocks.

Novel neural modulators.

Carbon monoxide neurotransmission activated by CK2 phosphorylation of heme oxygenase-2.

Cytochrome c binds to inositol (1,4,5) trisphosphate receptors, amplifying calcium-dependent apoptosis.

Apoptosis and calcium: new roles for cytochrome c and inositol 1,4,5-trisphosphate.

Agonist-induced Ca2+ entry determined by inositol 1,4,5-trisphosphate recognition.

Carbon monoxide mediates vasoactive intestinal polypeptide-associated nonadrenergic/noncholinergic neurotransmission.

Heme oxygenase-2 is activated by calcium-calmodulin.

Inositol 1,4,5-trisphosphate receptors as signal integrators.

A peptide inhibitor of cytochrome c/inositol 1,4,5-trisphosphate receptor binding blocks intrinsic and extrinsic cell death pathways.

Requirement of biphasic calcium release from the endoplasmic reticulum for Fas-mediated apoptosis.

Hepatitis C virus core protein increases mitochondrial ROS production by stimulation of Ca2+ uniporter activity.

Gene identification and evidence for expression of G protein alpha subunits, phospholipase C, and an inositol 1,4,5-trisphosphate receptor in Aplysia californica rhinophore.

Effect of insulin on the inflammatory and acute phase response after burn injury.

Requirement of inositol 1,4,5-trisphosphate receptors for tumor-mediated lymphocyte apoptosis.

Characterization of the inflammatory response during acute and post-acute phases after severe burn.

Role of heme oxygenase-2 in pial arteriolar response to acetylcholine in mice with and without transfusion of cell-free hemoglobin polymers.

Generation of spinal motor neurons from human fetal brain-derived neural stem cells: role of basic fibroblast growth factor.

Severe burn-induced endoplasmic reticulum stress and hepatic damage in mice.

Calcium and ER stress mediate hepatic apoptosis after burn injury.

T-cell receptor complex is essential for Fas signal transduction.

FAD mutations in amyloid precursor protein do not directly perturb intracellular calcium homeostasis.

Insulin protects against hepatic damage postburn.

Endoplasmic reticulum stress and insulin resistance post-trauma: similarities to type 2 diabetes.

Ubiquilin-1 is a molecular chaperone for the amyloid precursor protein.

Post-burn hepatic insulin resistance is associated with endoplasmic reticulum (ER) stress.

Severe injury is associated with insulin resistance, endoplasmic reticulum stress response, and unfolded protein response.

Thermal Injury Activates eEF2k-Dependent eEF2 Pathway in Pediatric Patients.

Measurement of Hepatic Protein Fractional Synthetic Rate with Stable Isotope Labeling Technique in Thapsigargin Stressed HepG2 Cells.

Molecular architecture of the inositol 1,4,5-trisphosphate receptor pore.

Propranolol improves impaired hepatic phosphatidylinositol 3-kinase/akt signaling after burn injury.

Structure-Function Of The Tumor Suppressor BRCA1.

Ubiquilin-1 regulates amyloid precursor protein maturation and degradation by stimulating K63-linked polyubiquitination of lysine 688.