Frann Antignano

Re: the terminology issue for myeloid-derived suppressor cells.

The role of SHIP in macrophages.

SHIP prevents lipopolysaccharide from triggering an antiviral response in mice.

Bone marrow-derived mast cells accumulate in the central nervous system during inflammation but are dispensable for experimental autoimmune encephalomyelitis pathogenesis.

SHIP1 is a repressor of mast cell hyperplasia, cytokine production, and allergic inflammation in vivo.

Prion protein expression and release by mast cells after activation.

SHIP represses the generation of IL-3-induced M2 macrophages by inhibiting IL-4 production from basophils.

SHIP is required for dendritic cell maturation.

Tyrosine phosphorylation of SHIP promotes its proteasomal degradation.

Activating and inhibitory functions for the histone lysine methyltransferase G9a in T helper cell differentiation and function.

SHIP negatively regulates Flt3L-derived dendritic cell generation and positively regulates MyD88-independent TLR-induced maturation.

TLR agonists that induce IFN-beta abrogate resident macrophage suppression of T cells.

Role of SHIP in cancer.

SHIP represses Th2 skewing by inhibiting IL-4 production from basophils.

MyD88-dependent SHIP1 regulates proinflammatory signaling pathways in dendritic cells after monophosphoryl lipid A stimulation of TLR4.

Assessing the role of CD103 in immunity to an intestinal helminth parasite.

SHIP-deficient dendritic cells, unlike wild type dendritic cells, suppress T cell proliferation via a nitric oxide-independent mechanism.