Xiaorui Shi

The influence of loud sound stress on expression of osmotic stress protein 94 in the murine inner ear.

The cochlear pericytes.

The influence of NF-kappaB signal-transduction pathways on the murine inner ear by acoustic overstimulation.

Cochlear pericyte responses to acoustic trauma and the involvement of hypoxia-inducible factor-1alpha and vascular endothelial growth factor.

Visualization and contractile activity of cochlear pericytes in the capillaries of the spiral ligament.

Volumetric Imaging of Blood Flow within Cochlea in Gerbil in vivo.

Volumetric in vivo imaging of microvascular perfusion within the intact cochlea in mice using ultra-high sensitive optical microangiography.

Resident macrophages in the cochlear blood-labyrinth barrier and their renewal via migration of bone-marrow-derived cells.

Bone marrow cell recruitment mediated by inducible nitric oxide synthase/stromal cell-derived factor-1alpha signaling repairs the acoustically damaged cochlear blood-labyrinth barrier.

Na+/K+-ATPase α1 identified as an abundant protein in the blood-labyrinth barrier that plays an essential role in the barrier integrity.

A differentially amplified motion in the ear for near-threshold sound detection.

Fibro-vascular coupling in the control of cochlear blood flow.

Lactate dilates cochlear capillaries via type V fibrocyte-vessel coupling signaled by nNOS.

Physiopathology of the cochlear microcirculation.

Perivascular-resident macrophage-like melanocytes in the inner ear are essential for the integrity of the intrastrial fluid-blood barrier.

Endothelial cell, pericyte, and perivascular resident macrophage-type melanocyte interactions regulate cochlear intrastrial fluid-blood barrier permeability.

Isolation and culture of endothelial cells, pericytes and perivascular resident macrophage-like melanocytes from the young mouse ear.

Perivascular macrophage-like melanocyte responsiveness to acoustic trauma--a salient feature of strial barrier associated hearing loss.

Characterization and inflammatory response of perivascular-resident macrophage-like melanocytes in the vestibular system.

Thin and open vessel windows for intra-vital fluorescence imaging of murine cochlear blood flow.

JAK2/STAT3 inhibition attenuates noise-induced hearing loss.

Structural changes in thestrial blood-labyrinth barrier of aged C57BL/6 mice.

Lipopolysaccharide-induced middle ear inflammation disrupts the cochlear intra-strial fluid-blood barrier through down-regulation of tight junction proteins.

Pathophysiology of the cochlear intrastrial fluid-blood barrier (review).

Culture media-based selection of endothelial cells, pericytes, and perivascular-resident macrophage-like melanocytes from the young mouse vestibular system.

Platelet-Derived Growth Factor Subunit B Signaling Promotes Pericyte Migration in Response to Loud Sound in the Cochlear Stria Vascularis.

Microvascular networks in the area of the auditory peripheral nervous system.

Delivery of therapeutics to the inner ear: The challenge of the blood-labyrinth barrier.

Vascular regeneration in adult mouse cochlea stimulated by VEGF-A and driven by NG2-derived cells ex vivo.

Acoustic Trauma Causes Cochlear Pericyte-to-Myofibroblast-Like Cell Transformation and Vascular Degeneration, and Transplantation of New Pericytes Prevents Vascular Atrophy.

Suppression of Connexin 43 Leads to Strial Vascular Hyper-Permeability, Decrease in Endocochlear Potential, and Mild Hearing Loss.

VEGFA165 gene therapy ameliorates blood-labyrinth barrier breakdown and hearing loss.