Ionita Ghiran

Complement receptor 1/CD35 is a receptor for mannan-binding lectin.

Expression and function of C1q receptors and C1q binding proteins at the cell surface.

Calreticulin is at the surface of circulating neutrophils and uses CD59 as an adaptor molecule.

A role for lipid rafts in C1q-triggered O2- generation by human neutrophils.

Phagocytosis of Salmonella montevideo by human neutrophils: immune adherence increases phagocytosis, whereas the bacterial surface determines the route of intracellular processing.

Human eosinophils secrete preformed, granule-stored interleukin-4 through distinct vesicular compartments.

Confocal light absorption and scattering spectroscopic microscopy monitors organelles in live cells with no exogenous labels.

Airway eosinophils: allergic inflammation recruited professional antigen-presenting cells.

Ligation of erythrocyte CR1 induces its clustering in complex with scaffolding protein FAP-1.

Complex dynamics of human red blood cell flickering: alterations with in vivo aging.

Eosinophil granules function extracellularly as receptor-mediated secretory organelles.

C3b deposition on human erythrocytes induces the formation of a membrane skeleton-linked protein complex.

Light-scattering spectroscopy differentiates fetal from adult nucleated red blood cells: may lead to noninvasive prenatal diagnosis.

Helicobacter pylori moves through mucus by reducing mucin viscoelasticity.

Complement receptor 1 expression on mouse erythrocytes mediates clearance of Streptococcus pneumoniae by immune adherence.

Ligation of complement receptor 1 increases erythrocyte membrane deformability.

Examining the role of microvesicles to develop prognostic and diagnostic assays.

Introduction to fluorescence microscopy.

Systemic lupus erythematosus serum deposits C4d on red blood cells, decreases red blood cell membrane deformability, and promotes nitric oxide production.

Eosinophil extracellular DNA trap cell death mediates lytic release of free secretion-competent eosinophil granules in humans.

Human complement receptor type 1/CD35 is an Epstein-Barr Virus receptor.

Malaria-infected erythrocyte-derived microvesicles mediate cellular communication within the parasite population and with the host immune system.

CR1-mediated ATP release by human red blood cells promotes CR1 clustering and modulates the immune transfer process.

C4d deposits on the surface of RBCs in trauma patients and interferes with their function.

Bio-inspired cryo-ink preserves red blood cell phenotype and function during nanoliter vitrification.

NAD+ protects against EAE by regulating CD4+ T-cell differentiation.

Dynamic actin filaments control the mechanical behavior of the human red blood cell membrane.

Levitational Image Cytometry with Temporal Resolution.

Magnetic levitation of single cells.

Smart-Phone Based Magnetic Levitation for Measuring Densities.

Potential functional applications of extracellular vesicles: a report by the NIH Common Fund Extracellular RNA Communication Consortium.

Diurnal Variations of Circulating Extracellular Vesicles Measured by Nano Flow Cytometry.

Ligation of Glycophorin A Generates Reactive Oxygen Species Leading to Decreased Red Blood Cell Function.

NAD(+) regulates Treg cell fate and promotes allograft survival via a systemic IL-10 production that is CD4(+) CD25(+) Foxp3(+) T cells independent.

Detection and quantification of subtle changes in red blood cell density using a cell phone.

Infected erythrocyte-derived extracellular vesicles alter vascular function via regulatory Ago2-miRNA complexes in malaria.

Extracellular Microvesicle Production by Human Eosinophils Activated by "Inflammatory" Stimuli.

DDiT4L promotes autophagy and inhibits pathological cardiac hypertrophy in response to stress.

Exercise-induced circulating extracellular vesicles protect against cardiac ischemia-reperfusion injury.

Red Blood Cells: A Source of Extracellular Vesicles.

Detection of membrane-bound and soluble antigens by magnetic levitation.

Small RNA-seq during acute maximal exercise reveal RNAs involved in vascular inflammation and cardiometabolic health: brief report.

Malaria infected red blood cells release small regulatory RNAs through extracellular vesicles.

Mast cells regulate CD4 T-cell differentiation in the absence of antigen presentation.

A Novel Implementation of Magnetic Levitation to Quantify Leukocyte Size, Morphology, and Magnetic Properties to Identify Patients With Sepsis.

Myosin IIA interacts with the spectrin-actin membrane skeleton to control red blood cell membrane curvature and deformability.

Comprehensive multi-center assessment of small RNA-seq methods for quantitative miRNA profiling.

High-fidelity detection and sorting of nanoscale vesicles in viral disease and cancer.

Correction to: Exercise-induced circulating extracellular vesicles protect against cardiac ischemia-reperfusion injury.

Characterization of plasma-derived protoporphyrin-IX-positive extracellular vesicles following 5-ALA use in patients with malignant glioma.

Rational Design of a Bifunctional AND-Gate Ligand To Modulate Cell-Cell Interactions.

Detection of Extracellular Vesicle RNA Using Molecular Beacons.

MIFlowCyt-EV: a framework for standardized reporting of extracellular vesicle flow cytometry experiments.

Profiling Extracellular Long RNA Transcriptome in Human Plasma and Extracellular Vesicles for Biomarker Discovery.

VWF maturation and release are controlled by 2 regulators of Weibel-Palade body biogenesis: exocyst and BLOC-2.

Discovery and Verification of Extracellular miRNA Biomarkers for Non-invasive Prediction of Pre-eclampsia in Asymptomatic Women.

Measurement and standardization challenges for extracellular vesicle therapeutic delivery vectors.

Mir-30d Regulates Cardiac Remodeling by Intracellular and Paracrine Signaling.

High-Sensitivity Glycan Profiling of Blood-Derived Immunoglobulin G, Plasma, and Extracellular Vesicle Isolates with Capillary Zone Electrophoresis-Mass Spectrometry.

A simple, high-throughput method of protein and label removal from extracellular vesicle samples.

The NIH Somatic Cell Genome Editing program.