Hilde Gillijns

Ventricular phosphodiesterase-5 expression is increased in patients with advanced heart failure and contributes to adverse ventricular remodeling after myocardial infarction in mice.

Increased cardiac myocyte PDE5 levels in human and murine pressure overload hypertrophy contribute to adverse LV remodeling.

Placental growth factor 2--A potential therapeutic strategy for chronic myocardial ischemia.

Concomitant Phosphodiesterase 5 Inhibition Enhances Myocardial Protection by Inhaled Nitric Oxide in Ischemia-Reperfusion Injury.

Neovascularization Potential of Blood Outgrowth Endothelial Cells From Patients With Stable Ischemic Heart Failure Is Preserved.

Right ventricular and pulmonary vascular reserve in asymptomatic BMPR2 mutation carriers.

Decreased Soluble Guanylate Cyclase Contributes to Cardiac Dysfunction Induced by Chronic Doxorubicin Treatment in Mice.

Sustained Placental Growth Factor-2 Treatment Does Not Aggravate Advanced Atherosclerosis in Ischemic Cardiomyopathy.

Molecular signature of progenitor cells isolated from young and adult human hearts.

C-reactive protein during and after myocardial infarction in relation to cardiac injury and left ventricular function at follow-up.

Peripheral Blood RNA Levels of and Are New Independent Predictors of Left Ventricular Dysfunction After Acute Myocardial Infarction.

Cardiac Microvascular Endothelial Cells in Pressure Overload-Induced Heart Disease.

Clec4e-Receptor Signaling in Myocardial Repair After Ischemia-Reperfusion Injury.

Arteriovenous Fistulae in Chronic Kidney Disease and the Heart: Physiological, Histological, and Transcriptomic Characterization of a Novel Rat Model.

Alginate sulfate-nanoparticles loaded with hepatocyte growth factor and insulin-like growth factor-1 improve left ventricular repair in a porcine model of myocardial ischemia reperfusion injury.