Ilona Jaspers

Role of oxidative stress on diesel-enhanced influenza infection in mice.

Role of Ras in metal-induced EGF receptor signaling and NF-kappaB activation in human airway epithelial cells.

Oxidative stress activates anion exchange protein 2 and AP-1 in airway epithelial cells.

Transcription factor activation following exposure of an intact lung preparation to metallic particulate matter.

The role of biomethylation in toxicity and carcinogenicity of arsenic: a research update.

Differential activation of AP-1 in human bladder epithelial cells by inorganic and methylated arsenicals.

Stress-responsive JNK mitogen-activated protein kinase mediates aspirin-induced suppression of B16 melanoma cellular proliferation.

Superoxide-dependent iron uptake: a new role for anion exchange protein 2.

Photochemical products in urban mixtures enhance inflammatory responses in lung cells.

Effects of 1,3-butadiene, isoprene, and their photochemical degradation products on human lung cells.

Ultrafine carbon particles induce interleukin-8 gene transcription and p38 MAPK activation in normal human bronchial epithelial cells.

Diesel exhaust enhances influenza virus infections in respiratory epithelial cells.

Oxidant generation promotes iron sequestration in BEAS-2B cells exposed to asbestos.

Diesel exhaust enhances virus- and poly(I:C)-induced Toll-like receptor 3 expression and signaling in respiratory epithelial cells.

Diesel exhaust particle-exposed human bronchial epithelial cells induce dendritic cell maturation.

Atmospheric photochemical transformations enhance 1,3-butadiene-induced inflammatory responses in human epithelial cells: The role of ozone and other photochemical degradation products.

Zn2+-induced NF-kappaB-dependent transcriptional activity involves site-specific p65/RelA phosphorylation.

Diesel exhaust enhanced susceptibility to influenza infection is associated with decreased surfactant protein expression.

Air pollution and respiratory viral infection.

Modulation of pulmonary inflammatory responses and antimicrobial defenses in mice exposed to diesel exhaust.

Mechanisms of LPS-induced CD40 expression in human peripheral blood monocytic cells.

Localization of type I interferon receptor limits interferon-induced TLR3 in epithelial cells.

Increased nasal epithelial ciliary beat frequency associated with lifestyle tobacco smoke exposure.

Exacerbation of allergic inflammation in mice exposed to diesel exhaust particles prior to viral infection.

Reduced expression of IRF7 in nasal epithelial cells from smokers after infection with influenza.

Disruption of microRNA expression in human airway cells by diesel exhaust particles is linked to tumorigenesis-associated pathways.

Phenotypic and physiologic variability in nasal epithelium cultured from smokers and non-smokers exposed to secondhand tobacco smoke.

The NRF2-mediated oxidative stress response pathway is associated with tumor cell resistance to arsenic trioxide across the NCI-60 panel.

Tobacco smoke exposure and altered nasal responses to live attenuated influenza virus.

Epithelial cells from smokers modify dendritic cell responses in the context of influenza infection.

Epigenetic changes induced by air toxics: formaldehyde exposure alters miRNA expression profiles in human lung cells.

Nrf2 expression modifies influenza A entry and replication in nasal epithelial cells.

GSTM1 modulation of IL-8 expression in human bronchial epithelial cells exposed to ozone.

A toxicogenomic comparison of primary and photochemically altered air pollutant mixtures.

Nasal lavage natural killer cell function is suppressed in smokers after live attenuated influenza virus.

Diesel exhaust exposure and nasal response to attenuated influenza in normal and allergic volunteers.

Alteration of the nasal responses to influenza virus by tobacco smoke.

Effects of air pollutants on innate immunity: the role of Toll-like receptors and nucleotide-binding oligomerization domain-like receptors.

Live attenuated influenza virus (LAIV) induces different mucosal T cell function in nonsmokers and smokers.

Exposure to ozone modulates human airway protease/antiprotease balance contributing to increased influenza A infection.

In vitro exposures in diesel exhaust atmospheres: resuspension of PM from filters versus direct deposition of PM from air.

Epithelial cells, the "switchboard" of respiratory immune defense responses: effects of air pollutants.

Airway protease/antiprotease imbalance in atopic asthmatics contributes to increased influenza A virus cleavage and replication.

Influenza enhances caspase-1 in bronchial epithelial cells from asthmatic volunteers and is associated with pathogenesis.

Sulforaphane induces SLPI secretion in the nasal mucosa.

Ozone exposed epithelial cells modify cocultured natural killer cells.

Diesel exhaust particles modify natural killer cell function and cytokine release.

Bronchoscopy-Derived Correlates of Lung Injury following Inhalational Injuries: A Prospective Observational Study.

DNA methylation in nasal epithelial cells from smokers: identification of ULBP3-related effects.