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Center of Operative Medicine,
Department of Visceral,
Transplant and Thoracic Surgery,
Center of Operative Medicine, Department of Visceral, Transplant and Thoracic Surgery
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Impaired Endothelial Nitric Oxide Synthase Homodimer Formation Triggers Development of Transplant Vasculopathy - Insights from a Murine Aortic Transplantation Model.
Scientific reports Nov, 2016 | Pubmed ID: 27883078
Innsbruck Medical University
Medical University Innsbruck
Rupert Oberhuber*,1,
Benno Cardini*,1,
Markus Kofler1,
Paul Ritschl1,
Robert Oellinger1,
Felix Aigner1,
Robert Sucher1,
Stefan Schneeberger1,
Johann Pratschke1,
Gerald Brandacher2,
Manuel Maglione1
1Center of Operative Medicine, Department of Visceral, Transplant and Thoracic Surgery, Innsbruck Medical University,
2Department of Plastic and Reconstructive Surgery, Johns Hopkins University School of Medicine
Sven R Hein1,
Rebecca Eiter1,
Martin Hermann2,
Markus Kofler1,3,
Gerald Brandacher1,4,
1Center of Operative Medicine, Department of Visceral, Transplant and Thoracic Surgery, Medical University Innsbruck,
2Department of Anesthesiology and Critical Care Medicine, Medical University Innsbruck,
3Department of Cardiac Surgery, Medical University Innsbruck,
4Department of Plastic and Reconstructive Surgery, Vascularized Composite Allotransplantation (VCA) Laboratory, Johns Hopkins University School of Medicine
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