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DOI :
10.3791/63949-v
August 8th, 2022
Chapters
0:04
Introduction
0:35
Evaluation of the Cardiac Morphology and Function
1:54
Results: Validating the Pathogenicity of G823E Mutation Using the C57BL/6N Murine Model
3:17
Conclusion
基于我们临床工作中发现的家族性遗传性心肌病家族,我们通过CRISPR / Cas9介导的基因组工程在小鼠MYH7位点创建了一个具有点突变(G823E)的C57BL / 6N小鼠模型来验证该突变。
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