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Polymorphisms in the human xeroderma pigmentosum group A gene and their impact on cell survival and nucleotide excision repair.
DNA repair Jul, 2002 | Pubmed ID: 12509227
XP-A cells complemented with Arg228Gln and Val234Leu polymorphic XPA alleles repair BPDE-induced DNA damage better than cells complemented with the wild type allele.
DNA repair Mar, 2005 | Pubmed ID: 15661657
Telomerase-immortalized human fibroblasts retain UV-induced mutagenesis and p53-mediated DNA damage responses.
DNA repair Jan, 2006 | Pubmed ID: 16140041
Comment on "Carbon-negative biofuels from low-input high-diversity grassland biomass".
Science (New York, N.Y.) Jun, 2007 | Pubmed ID: 17569846
Predicting diabetic ketoacidosis in children by measuring end-tidal CO2 via non-invasive nasal capnography.
Journal of paediatrics and child health Oct, 2007 | Pubmed ID: 17854452
Human rhinovirus proteinase 2A induces TH1 and TH2 immunity in patients with chronic obstructive pulmonary disease.
The Journal of allergy and clinical immunology Jun, 2010 | Pubmed ID: 20430426
Respiratory tract allergic disease and atopy: experimental evidence for a fungal infectious etiology.
Medical mycology : official publication of the International Society for Human and Animal Mycology Apr, 2011 | Pubmed ID: 20807032
Seeking common pathophysiology in asthma, atopy and sinusitis.
Trends in immunology Feb, 2011 | Pubmed ID: 21239229
Proteinases as molecular adjuvants in allergic airway disease.
Biochimica et biophysica acta Nov, 2011 | Pubmed ID: 21712069
Clonidine in cyclic vomiting.
Journal of pediatric gastroenterology and nutrition Aug, 2011 | Pubmed ID: 21788767
Fungal chitin from asthma-associated home environments induces eosinophilic lung infiltration.
Journal of immunology (Baltimore, Md. : 1950) Sep, 2011 | Pubmed ID: 21824866
Necessary and sufficient role for T helper cells to prevent fungal dissemination in allergic lung disease.
Infection and immunity Nov, 2011 | Pubmed ID: 21875960
The laryngeal saccule: Clinical significance.
Clinical anatomy (New York, N.Y.) Dec, 2011 | Pubmed ID: 22162142
Tracheobronchial mycosis in a retrospective case-series study of five status asthmaticus patients.
Clinical immunology (Orlando, Fla.) Feb, 2013 | Pubmed ID: 23280490
IL-33-responsive innate lymphoid cells are an important source of IL-13 in chronic rhinosinusitis with nasal polyps.
American journal of respiratory and critical care medicine Aug, 2013 | Pubmed ID: 23805875
Agonistic induction of PPARγ reverses cigarette smoke-induced emphysema.
The Journal of clinical investigation Mar, 2014 | Pubmed ID: 24569375
Blocking KV1.3 channels inhibits Th2 lymphocyte function and treats a rat model of asthma.
The Journal of biological chemistry May, 2014 | Pubmed ID: 24644290
Airway surface mycosis in chronic TH2-associated airway disease.
The Journal of allergy and clinical immunology Aug, 2014 | Pubmed ID: 24928648
Airway fibrinogenolysis and the initiation of allergic inflammation.
Annals of the American Thoracic Society Dec, 2014 | Pubmed ID: 25525732
Long-Acting Beta Agonists Enhance Allergic Airway Disease.
PloS one , 2015 | Pubmed ID: 26605551
Allergen-encoded signals that control allergic responses.
Current opinion in allergy and clinical immunology Feb, 2016 | Pubmed ID: 26658015
Prevalence of confirmed asthma varies in chronic rhinosinusitis subtypes.
International forum of allergy & rhinology Apr, 2016 | Pubmed ID: 26678021
Preferential uptake of antioxidant carbon nanoparticles by T lymphocytes for immunomodulation.
Scientific reports Sep, 2016 | Pubmed ID: 27654170
Benefits of antifungal therapy in asthma patients with airway mycosis: A retrospective cohort analysis.
Immunity, inflammation and disease 06, 2018 | Pubmed ID: 29575717
Larval Infection and Lung Invasion Directly Induce Severe Allergic Airway Disease in Mice.
Infection and immunity 12, 2018 | Pubmed ID: 30249744
Selective cleavage of fibrinogen by diverse proteinases initiates innate allergic and antifungal immunity through CD11b.
The Journal of biological chemistry 05, 2019 | Pubmed ID: 30992366
Electronic cigarettes disrupt lung lipid homeostasis and innate immunity independent of nicotine.
The Journal of clinical investigation 10, 2019 | Pubmed ID: 31483291
Daniel E. Morales-Mantilla1,2,
Xinyan Huang3,7,
Philip Erice1,3,
Paul Porter4,
Yun Zhang1,5,
Mary Figueroa6,
Joya Chandra6,
Katherine Y. King2,
Farrah Kheradmand4,8,
Antony Rodríguez3,8
1Program in Immunology, Baylor College of Medicine,
2Department of Pediatrics, Section of Infectious Diseases, Baylor College of Medicine,
3Department of Medicine - Immunology Allergy and Rheumatology, Baylor College of Medicine,
4Department of Medicine, Pulmonary, Critical Care, Sleep Medicine, Baylor College of Medicine,
5Department of Pathology and Immunology, Baylor College of Medicine,
6Department of Pediatrics, Research and Department of Epigenetics and Molecular Carcinogenesis, The University of Texas MD Anderson Cancer Center,
7Division of Pulmonary and Critical Care Medicine, The First Affiliated Hospital of Sun Yat-sen University,
8Center for Translational Research on Inflammatory Diseases (CTRID), Michael E. DeBakey VA Medical Center
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