Kip M. Connor

Mitochondrial H2O2 regulates the angiogenic phenotype via PTEN oxidation.

A radically twisted lipid regulates vascular death.

Redox-dependent matrix metalloproteinase-1 expression is regulated by JNK through Ets and AP-1 promoter motifs.

Manganese superoxide dismutase protects from TNF-alpha-induced apoptosis by increasing the steady-state production of H2O2.

IGFBP3 suppresses retinopathy through suppression of oxygen-induced vessel loss and promotion of vascular regrowth.

Increased dietary intake of omega-3-polyunsaturated fatty acids reduces pathological retinal angiogenesis.

Overstaying their welcome: defective CX3CR1 microglia eyed in macular degeneration.

Manganese superoxide dismutase enhances the invasive and migratory activity of tumor cells.

Erythropoietin deficiency decreases vascular stability in mice.

Suppression of retinal neovascularization by erythropoietin siRNA in a mouse model of proliferative retinopathy.

Quantification and localization of the IGF/insulin system expression in retinal blood vessels and neurons during oxygen-induced retinopathy in mice.

A mechanosensitive transcriptional mechanism that controls angiogenesis.

Quantification of oxygen-induced retinopathy in the mouse: a model of vessel loss, vessel regrowth and pathological angiogenesis.

The mouse retina as an angiogenesis model.

Short communication: PPAR gamma mediates a direct antiangiogenic effect of omega 3-PUFAs in proliferative retinopathy.

5-Lipoxygenase metabolite 4-HDHA is a mediator of the antiangiogenic effect of ω-3 polyunsaturated fatty acids.

Complement involvement in neovascular ocular diseases.

PGC-1α regulates normal and pathological angiogenesis in the retina.

Melanocyte-secreted fibromodulin promotes an angiogenic microenvironment.

EGF-like-domain-7 is required for VEGF-induced Akt/ERK activation and vascular tube formation in an ex vivo angiogenesis assay.

The alternative complement pathway regulates pathological angiogenesis in the retina.

Strain difference in photoreceptor cell death after retinal detachment in mice.

Cytochrome P450-generated metabolites derived from ω-3 fatty acids attenuate neovascularization.

Characterization of a spontaneous retinal neovascular mouse model.