Jeejabai Radhakrishnan

Direct injection and expression in vivo of full-length cDNA of the cardiac isoform of alpha-2 macroglobulin induces cardiac hypertrophy in the rat heart.

Delineation of sequences essential for specific promoter activation during pressure overloaded hypertrophy or factor-induced hypertrophy.

Evaluation of the cardiac isoform of alpha2-macroglobulin as a factor inducing cardiac hypertrophy.

Circulating levels of cytochrome c after resuscitation from cardiac arrest: a marker of mitochondrial injury and predictor of survival.

Limiting sarcolemmal Na+ entry during resuscitation from ventricular fibrillation prevents excess mitochondrial Ca2+ accumulation and attenuates myocardial injury.

Zoniporide preserves left ventricular compliance during ventricular fibrillation and minimizes postresuscitation myocardial dysfunction through benefits on energy metabolism.

Activation of caspase-3 may not contribute to postresuscitation myocardial dysfunction.

Targeting mitochondria for resuscitation from cardiac arrest.

AVE4454B--a novel sodium-hydrogen exchanger isoform-1 inhibitor--compared less effective than cariporide for resuscitation from cardiac arrest.

Clinically plausible hyperventilation does not exert adverse hemodynamic effects during CPR but markedly reduces end-tidal PCO₂.

Protecting mitochondrial bioenergetic function during resuscitation from cardiac arrest.

Vitamin C compromises cardiac resuscitability in a rat model of ventricular fibrillation.

Erythropoietin facilitates resuscitation from ventricular fibrillation by signaling protection of mitochondrial bioenergetic function in rats.

High-dose erythropoietin during cardiac resuscitation lessens postresuscitation myocardial stunning in swine.

Effects of intraosseous erythropoietin during hemorrhagic shock in swine.

Cyclophilin-D: a resident regulator of mitochondrial gene expression.