Erik J. Plautz

A squirrel monkey model of poststroke motor recovery.

Post-infarct cortical plasticity and behavioral recovery using concurrent cortical stimulation and rehabilitative training: a feasibility study in primates.

Dissociation of sensorimotor deficits after rostral versus caudal lesions in the primary motor cortex hand representation.

In search of the motor engram: motor map plasticity as a mechanism for encoding motor experience.

Topographically divergent and convergent connectivity between premotor and primary motor cortex.

Extensive cortical rewiring after brain injury.

Behavioral and neurophysiological effects of delayed training following a small ischemic infarct in primary motor cortex of squirrel monkeys.

Ipsilateral connections of the ventral premotor cortex in a new world primate.

VEGF protein associates to neurons in remote regions following cortical infarct.

Effects of small ischemic lesions in the primary motor cortex on neurophysiological organization in ventral premotor cortex.

A single injection of D-amphetamine facilitates improvements in motor training following a focal cortical infarct in squirrel monkeys.

Effects of a rostral motor cortex lesion on primary motor cortex hand representation topography in primates.

A stretch reflex in extraocular muscles of species purportedly lacking muscle spindles.

Neuronal HIF-1 alpha protein and VEGFR-2 immunoreactivity in functionally related motor areas following a focal M1 infarct.

Early and late changes in the distal forelimb representation of the supplementary motor area after injury to frontal motor areas in the squirrel monkey.

A novel device to measure power grip forces in squirrel monkeys.

Combination of NEP 1-40 treatment and motor training enhances behavioral recovery after a focal cortical infarct in rats.

Distal forelimb representations in primary motor cortex are redistributed after forelimb restriction: a longitudinal study in adult squirrel monkeys.

Repetitive hypoxic preconditioning induces an immunosuppressed B cell phenotype during endogenous protection from stroke.