Joseph M. Reynolds

The fatty acid-binding protein, aP2, coordinates macrophage cholesterol trafficking and inflammatory activity. Macrophage expression of aP2 impacts peroxisome proliferator-activated receptor gamma and IkappaB kinase activities.

Deficiency of fatty acid-binding proteins in mice confers protection from development of experimental autoimmune encephalomyelitis.

Regulation of Th17 differentiation by epidermal fatty acid-binding protein.

MKP-1 is necessary for T cell activation and function.

Identification and characterization of a small molecule inhibitor of Fatty Acid binding proteins.

Smad3 differentially regulates the induction of regulatory and inflammatory T cell differentiation.

Toll-like receptor 2 signaling in CD4(+) T lymphocytes promotes T helper 17 responses and regulates the pathogenesis of autoimmune disease.

The E3 ubiquitin ligase GRAIL regulates T cell tolerance and regulatory T cell function by mediating T cell receptor-CD3 degradation.

Smad2 positively regulates the generation of Th17 cells.

IL-17 family member cytokines: regulation and function in innate immunity.

Follicular regulatory T cells expressing Foxp3 and Bcl-6 suppress germinal center reactions.

Interleukin-17C promotes Th17 cell responses and autoimmune disease via interleukin-17 receptor E.

Toll-like receptor 4 signaling in T cells promotes autoimmune inflammation.

Cutting edge: regulation of intestinal inflammation and barrier function by IL-17C.

Negative regulation of IL-17-mediated signaling and inflammation by the ubiquitin-specific protease USP25.

Toll-like receptor regulation of effector T lymphocyte function.

Epstein Barr virus-induced 3 (EBI3) together with IL-12 negatively regulates T helper 17-mediated immunity to Listeria monocytogenes infection.

Proatherogenic conditions promote autoimmune T helper 17 cell responses in vivo.

Editorial: the contribution of myeloid-derived suppression to inflammatory disease.

MAPK Phosphatase 7 Regulates T Cell Differentiation via Inhibiting ERK-Mediated IL-2 Expression.

Production of Anti-LPS IgM by B1a B Cells Depends on IL-1β and Is Protective against Lung Infection with Francisella tularensis LVS.

MAPK Phosphatase 5 Expression Induced by Influenza and Other RNA Virus Infection Negatively Regulates IRF3 Activation and Type I Interferon Response.