Sreedharan Sajikumar

Anisomycin inhibits the late maintenance of long-term depression in rat hippocampal slices in vitro.

Late-associativity, synaptic tagging, and the role of dopamine during LTP and LTD.

The type IV-specific phosphodiesterase inhibitor rolipram and its effect on hippocampal long-term potentiation and synaptic tagging.

Synaptic tagging and cross-tagging: the role of protein kinase Mzeta in maintaining long-term potentiation but not long-term depression.

Protein synthesis-dependent long-term functional plasticity: methods and techniques.

Mitogen-activated protein kinase-mediated reinforcement of hippocampal early long-term depression by the type IV-specific phosphodiesterase inhibitor rolipram and its effect on synaptic tagging.

Synergistic requirements for the induction of dopaminergic D1/D5-receptor-mediated LTP in hippocampal slices of rat CA1 in vitro.

Identification of compartment- and process-specific molecules required for "synaptic tagging" during long-term potentiation and long-term depression in hippocampal CA1.

Cognitive and emotional information processing: protein synthesis and gene expression.

Distinct single but not necessarily repeated tetanization is required to induce hippocampal late-LTP in the rat CA1.

PKM zeta maintains late long-term potentiation by N-ethylmaleimide-sensitive factor/GluR2-dependent trafficking of postsynaptic AMPA receptors.

Priming of short-term potentiation and synaptic tagging/capture mechanisms by ryanodine receptor activation in rat hippocampal CA1.

Protein kinase Mzeta is essential for the induction and maintenance of dopamine-induced long-term potentiation in apical CA1 dendrites.

Metaplasticity governs compartmentalization of synaptic tagging and capture through brain-derived neurotrophic factor (BDNF) and protein kinase Mzeta (PKMzeta).

Different compartments of apical CA1 dendrites have different plasticity thresholds for expressing synaptic tagging and capture.

Dopamine induces LTP differentially in apical and basal dendrites through BDNF and voltage-dependent calcium channels.

Making synapses strong: metaplasticity prolongs associativity of long-term memory by switching synaptic tag mechanisms.

Competition between recently potentiated synaptic inputs reveals a winner-take-all phase of synaptic tagging and capture.

Ubiquitin-Proteasome System Inhibition Promotes Long-Term Depression and Synaptic Tagging/Capture.

Lentiviral silencing of GSK-3β in adult dentate gyrus impairs contextual fear memory and synaptic plasticity.

Dopamine D1/D5 receptor signaling regulates synaptic cooperation and competition in hippocampal CA1 pyramidal neurons via sustained ERK1/2 activation.

Impaired spatial memory and enhanced long-term potentiation in mice with forebrain-specific ablation of the Stim genes.