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Department of Biology
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Answering the Call: Coping with DNA Damage at the Most Inopportune Time.
Journal of biomedicine & biotechnology , 2002 | Pubmed ID: 12488586
DNA damage-induced replication fork regression and processing in Escherichia coli.
Science (New York, N.Y.) Feb, 2003 | Pubmed ID: 12543983
RecA-dependent recovery of arrested DNA replication forks.
Annual review of genetics , 2003 | Pubmed ID: 14616075
RecO acts with RecF and RecR to protect and maintain replication forks blocked by UV-induced DNA damage in Escherichia coli.
The Journal of biological chemistry Jan, 2004 | Pubmed ID: 14625283
RuvAB and RecG are not essential for the recovery of DNA synthesis following UV-induced DNA damage in Escherichia coli.
Genetics Apr, 2004 | Pubmed ID: 15126385
When replication travels on damaged templates: bumps and blocks in the road.
Research in microbiology May, 2004 | Pubmed ID: 15142619
Recs preventing wrecks.
Mutation research Sep, 2005 | Pubmed ID: 16011837
Nucleotide excision repair or polymerase V-mediated lesion bypass can act to restore UV-arrested replication forks in Escherichia coli.
Journal of bacteriology Oct, 2005 | Pubmed ID: 16199565
Nascent DNA processing by RecJ favors lesion repair over translesion synthesis at arrested replication forks in Escherichia coli.
Proceedings of the National Academy of Sciences of the United States of America Jun, 2006 | Pubmed ID: 16754873
Monitoring DNA replication following UV-induced damage in Escherichia coli.
Methods in enzymology , 2006 | Pubmed ID: 16793416
RuvABC is required to resolve holliday junctions that accumulate following replication on damaged templates in Escherichia coli.
The Journal of biological chemistry Sep, 2006 | Pubmed ID: 16895921
Structural conservation of RecF and Rad50: implications for DNA recognition and RecF function.
The EMBO journal Feb, 2007 | Pubmed ID: 17255941
Inactivation of the DnaB helicase leads to the collapse and degradation of the replication fork: a comparison to UV-induced arrest.
Journal of bacteriology Aug, 2007 | Pubmed ID: 17526695
RecBCD and RecJ/RecQ initiate DNA degradation on distinct substrates in UV-irradiated Escherichia coli.
Radiation research Oct, 2007 | Pubmed ID: 17903041
RecA433 cells are defective in recF-mediated processing of disrupted replication forks but retain recBCD-mediated functions.
Mutation research Oct, 2008 | Pubmed ID: 18782580
Shifting replication between IInd, IIIrd, and IVth gears.
Proceedings of the National Academy of Sciences of the United States of America Apr, 2009 | Pubmed ID: 19357302
Nucleotide excision repair is a predominant mechanism for processing nitrofurazone-induced DNA damage in Escherichia coli.
Journal of bacteriology Aug, 2009 | Pubmed ID: 19465649
ATP binding, ATP hydrolysis, and protein dimerization are required for RecF to catalyze an early step in the processing and recovery of replication forks disrupted by DNA damage.
Journal of molecular biology Aug, 2010 | Pubmed ID: 20558179
Escherichia coli Fpg glycosylase is nonrendundant and required for the rapid global repair of oxidized purine and pyrimidine damage in vivo.
Journal of molecular biology Jul, 2011 | Pubmed ID: 21601577
Portland State University
H. Arthur Jeiranian*,1,
Brandy J. Schalow*,1,
Justin Courcelle1
1Department of Biology, Portland State University
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